This collection includes most of the ASU Theses and Dissertations from 2011 to present. ASU Theses and Dissertations are available in downloadable PDF format; however, a small percentage of items are under embargo. Information about the dissertations/theses includes degree information, committee members, an abstract, supporting data or media.

In addition to the electronic theses found in the ASU Digital Repository, ASU Theses and Dissertations can be found in the ASU Library Catalog.

Dissertations and Theses granted by Arizona State University are archived and made available through a joint effort of the ASU Graduate College and the ASU Libraries. For more information or questions about this collection contact or visit the Digital Repository ETD Library Guide or contact the ASU Graduate College at gradformat@asu.edu.

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Description
Attention-Deficit/Hyperactivity Disorder (ADHD) impacts 7% of children and is associated with serious impairment throughout the lifespan. Though considered a chronic, stable condition, symptoms fluctuate substantially during adolescence, and symptom trajectory is linked to adult outcomes. A small number of studies have examined symptom trajectory during adolescence, but these have predominately

Attention-Deficit/Hyperactivity Disorder (ADHD) impacts 7% of children and is associated with serious impairment throughout the lifespan. Though considered a chronic, stable condition, symptoms fluctuate substantially during adolescence, and symptom trajectory is linked to adult outcomes. A small number of studies have examined symptom trajectory during adolescence, but these have predominately examined demographic predictors. As a neurodevelopmental disorder, ADHD is theorized to arise from deficits in executive functions (EFs). Extant literature identifies three major components of EF- working memory, behavioral inhibition, and set shifting- as interrelated constructs underlying ADHD symptom expression. This study aimed to 1.) identify trajectories of ADHD symptoms, 2.) examine demographic predictors of trajectories, and 3.) examine whether EF predicts symptom trajectory using five timepoints from the Adolescent Brain Cognitive Development study, a large-scale, representative, population-based sample from the United States. 1,605 participants meeting criteria for ADHD included in analyses. ADHD symptoms were measured by parent report on the widely used Achenbach Child Behavior Checklist. Growth Mixture Modeling was used to model trajectories of ADHD symptoms. However, poor entropy indicated trajectories were not clearly differentiated and predictors could not be examined. Therefore, exploratory regression was conducted to examine predictors of ADHD symptom change from baseline to 3-year follow-up. Male sex, medication use, greater than high school parental education, and better EF all predicted increasing ADHD symptoms. Findings must be interpreted with caution due to their exploratory nature and poor validity of the measure used for ADHD symptoms, which was found to have sensitivity of only 22.58%. Given the strong theoretical and empirical link between ADHD symptoms and EF, additional research on the connection between EF and disorder trajectory with more robust measures of EF is warranted.
ContributorsChromik, Lindsay C (Author) / Friedman, Lauren (Thesis advisor) / McNeish, Daniel (Committee member) / Chassin, Laurie (Committee member) / Arizona State University (Publisher)
Created2024
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Description
Family disruption, or the separation of children from caregivers, has been well-established in prior literature as a risk factor for child maladjustment; however, little is known about how family disruption impacts youth into adulthood, particularly how it influences children’s later parenting of their own offspring. The present study examined whether

Family disruption, or the separation of children from caregivers, has been well-established in prior literature as a risk factor for child maladjustment; however, little is known about how family disruption impacts youth into adulthood, particularly how it influences children’s later parenting of their own offspring. The present study examined whether cumulative family disruption (i.e., parental hospitalization, death, incarceration, divorce) in childhood exerts effects on children’s parenting of their own offspring in adulthood, beyond other demographic characteristics and risk factors. Further, several potential mechanisms were hypothesized to underlie the association between family disruption in the first and second generation (G1-G2) family and later parenting provided from second-generation (G2) adults to third-generation (G3) children. Mediators included conflict and disorganization in the G1-G2 family and dysregulation in the G2 child.

Participants (N = 236 in models that included multiple G2 siblings; N = 110 in models without siblings) were drawn from a larger sample of at-risk (i.e., alcoholic) and comparison families followed longitudinally for over 30 years and across three generations. Four mediation models were estimated to examine effects of two separate G1-G2 family disruption components (deviance-related and health-related disruption) on parenting of G3, mediated by family conflict, family disorganization, and G2 dysregulation. Results indicated that health-related disruption impairs consistency of parenting provided to G3 offspring through conflict in the G1-G2 family. A direct effect of health-related disruption was also seen on parental monitoring. There were no direct or mediated effects of deviance-related disruption on parenting. Implications and future directions will be discussed.
ContributorsBlake, Austin Joy (Author) / Chassin, Laurie (Thesis advisor) / Meier, Madeline (Committee member) / Wolchik, Sharlene (Committee member) / McNeish, Daniel (Committee member) / Arizona State University (Publisher)
Created2019
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Description
Despite the established co-prevalence of substance use (SU) and disordered eating (DE), few longitudinal studies have sought to examine their shared development. Findings have been inconsistent within the extant literature. This may be attributable in part to several methodological aspects, including overlooking distinct psychopharmacological properties of common substances of abuse,

Despite the established co-prevalence of substance use (SU) and disordered eating (DE), few longitudinal studies have sought to examine their shared development. Findings have been inconsistent within the extant literature. This may be attributable in part to several methodological aspects, including overlooking distinct psychopharmacological properties of common substances of abuse, examining only between-person relations, and failing to account for shared risk factors. The current study sought to address these gaps by applying latent curve models with structured residuals (LCM-SR) to a preexisting, national sample of adolescent girls followed into adulthood, Add Health. In Aim 1, between-person effects examined the simultaneous development of alcohol, tobacco, and marijuana use and DE behaviors in substance-specific models. In Aim 2, bivariate latent curve models were expanded to account for within-person effects (LCM-SR) in order to examine the potentially bidirectional, prospective relationship between use of a specific substance and DE. Lastly, models accounted for shared developmental risk factors. Findings of the current study demonstrate preliminary evidence of substance-specific effects with DE emerging in adolescence. Across model-building steps, DE engagement in early adolescence was significantly associated with growth in tobacco use and marginally associated with marijuana use. Appetitive side-effects of both substances may link use with DE behaviors and enhance instrumental use for weight control. Significant associations did not emerge between alcohol and DE, and results of the conditional model indicate this co-occurrence is best explained by third variable mechanisms. Implications for prevention are discussed.
ContributorsBruening, Amanda B (Author) / Corbin, William (Thesis advisor) / Chassin, Laurie (Committee member) / Meier, Madeline (Committee member) / McNeish, Daniel (Committee member) / Arizona State University (Publisher)
Created2021
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Description
Negative Urgency and Positive Urgency are important subfacets of a propensity to rash action. There is currently limited research on parental antecedents of Negative Urgency and Positive Urgency. The current study investigated whether parent personality and parenting behaviors predict adolescent Negative Urgency and Positive Urgency. Data were taken from a

Negative Urgency and Positive Urgency are important subfacets of a propensity to rash action. There is currently limited research on parental antecedents of Negative Urgency and Positive Urgency. The current study investigated whether parent personality and parenting behaviors predict adolescent Negative Urgency and Positive Urgency. Data were taken from a community sample with parent personality, positive parenting behaviors, and child Negative Urgency and Positive Urgency measured at separate timepoints. Structural equation models were used to examine whether parent personality predicted adolescent Negative Urgency and Positive Urgency and whether positive parenting mediated this relationship. There was no evidence for a relationship between parent personality and children’s Negative Urgency and Positive Urgency. In addition, there was no relationship between parenting behaviors and child Negative and Positive Urgency in cross-reporter models, but child-reported parenting predicted later adolescent-reported Negative and Positive Urgency. Greater positive parenting, as perceived by children, was related to less Negative and Positive Urgency when they were adolescents. More research is needed to understand whether the current results are due to reporter bias or whether child-perceived parenting behaviors influence the development of adolescent Negative and Positive Urgency.
ContributorsBui, Leena (Author) / Chassin, Laurie (Thesis advisor) / Corbin, William (Committee member) / Lemery-Chalfant, Kathryn (Committee member) / McNeish, Daniel (Committee member) / Arizona State University (Publisher)
Created2022