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Description
Family adaptation to child developmental disability is a dynamic transactional process that has yet to be tested in a longitudinal, rigorous fashion. In addition, although children with developmental delays frequently have behavior problems, not enough research has examined possible underlying mechanisms in the relation between child developmental delay, adaptation and behavior problems. In the current study, factor analysis examined how best to conceptualize the construct of family adaptation to developmental delay. Also, longitudinal growth curve modeling tested models in which child behavior problems mediated the relation between developmental risk and indices of family adaptation. Participants included 130 typically developing children and their families (Mental Development Index [MDI] > 85) and 104 children with developmental delays and their families (MDI < 85). Data were collected yearly between the ages of three and eight as part of a multi-site, longitudinal investigation examining the interrelations among children's developmental status, family processes, and the emergence of child psychopathology. Results of the current study indicated that adaptation is best conceptualized as a multi-index construct. Different aspects of adaptation changed in unique ways over time, with some facets of adaptation remaining stable while others fluctuated. Child internalizing and externalizing behavior problems were found to decrease over time for both children with developmental delays and typically developing children. Child behavior problems were also found to mediate the relation between developmental risk and family adaptation for over half of the mediation pathways. Significant mediation results indicated that children with developmental delays showed higher early levels of behavior problems, which in turn was associated with more maladaptive adaptation. These findings provide further evidence that families of children with developmental delays experience both positive and more challenging changes in their families over time. This study implies important next steps for research and clinical practice in the area of developmental disability.
ContributorsPedersen y Arbona, Anita (Author) / Crnic, Keith A (Thesis advisor) / Sandler, Irwin (Committee member) / Lemery, Kathryn (Committee member) / Enders, Craig (Committee member) / Arizona State University (Publisher)
Created2011
Description
The purpose of the current study was to use structural equation modeling-based quantitative genetic models to characterize latent genetic and environmental influences on proneness to three discrete negative emotions in middle childhood, according to mother-report, father-report and in-home observation. One primary aim was to test the extent to which covariance among the three emotions could be accounted for by a single, common genetically- and environmentally-influenced negative emotionality factor. A second aim was to examine the extent to which different reporters appeared to be tapping into the same genetically- and environmentally-influenced aspects of each emotion. According to mother- and father-report, moderate to high genetic influences were evident for all emotions, with mother- and father-report of fear and father-report of anger showing the highest heritability. Significant common environmental influences were also found for mother-report of anger and sadness in both univariate and multivariate models. For observed emotion, anger was moderately heritable with no evidence for common environmental variance, but sadness, object fear and social fear all showed modest to moderate common environmental influences and no significant genetic variance. In addition, cholesky decompositions examining genetic and environmental influences across reporter suggested that despite considerable overlap between mother-report and father-report, there was also reporter-specific variance on anger, sadness, and fear. Specifically, there were significant common environmental influences on mother-report of anger- and sadness that were not shared with father-report, and genetic influences on father-report of sadness and fear that were not shared with mother-report. In-home observations were not highly correlated enough with parent-report to support multivariate analysis for any emotion. Finally, according to both mother- and father-report, a single set of genetic and environmental influences was sufficient to account for covariance among all three negative emotions. However, fear was primarily explained by genetic influences not shared with other emotions, and anger also showed considerable emotion-specific genetic variance. In both cases, findings support the value of a more emotion-specific approach to temperament, and highlight the need to consider distinctions as well as commonalities across emotions, reporters and situations.
ContributorsClifford, Sierra (Author) / Lemery, Kathryn (Thesis advisor) / Shiota, Michelle (Committee member) / Eisenberg, Nancy (Committee member) / Arizona State University (Publisher)
Created2013
Description
Using a sample of children from divorced homes, the current study assesses the effects of family relationship variables on romantic outcomes in young adulthood, through the influence of several individual-level variables. In particular, children's coping efficacy and peer competence are examined as mediators of the effects of parenting and interparental conflict on children's later romantic involvement and relationship quality. Assessments occurred during childhood, when children were between the ages of nine and 12, in adolescence, when children were ages 15 to 18, and in young adulthood, when children were ages 24 to 27, spanning a period of 15 years. Childhood and adolescent variables were measured using child- and mother-report data and young adult measures were completed by the young adults and their romantic partners. One model was tested using all participants in the sample, regardless of whether they were romantically involved in young adulthood, and revealed that maternal warmth in childhood was linked with children's coping efficacy six years later, which was marginally related to an increased likelihood of being romantically involved and to decreased romantic attachment at the 15-year follow-up. A model with only the participants who were romantically involved in young adulthood also revealed a link between childhood maternal warmth and coping efficacy in adolescence, which was then marginally related to increased romantic satisfaction and to confidence in the romantic relationship in young adulthood. Marginal mediation was also found for several of the proposed paths, and there was little evidence to support path differences between males and females. Implications of the present findings for research with children from divorced families and the development of preventive interventions are discussed. In particular, parenting, interparental conflict, peer competence, and coping efficacy are examined as modifiable targets for change and existing preventive interventions employing these targets are described.
ContributorsSigal, Amanda (Author) / Sandler, Irwin N. (Thesis advisor) / Millsap, Roger (Committee member) / Crnic, Keith (Committee member) / Lemery, Kathryn (Committee member) / Arizona State University (Publisher)
Created2012
Description
Data from the NICHD Study of Early Child Care and Youth Development (SECCYD) was used to study the role of child individual, parental, and environmental predictors of anxiety across childhood and adolescence. Longitudinal growth modeling was used to examine the influence of behavioral inhibition, parental control, parental anxiety and stressful life events on the developmental progression of anxiety from 4 to 15 years of age. Based on these data, it appears that there are significant developmental differences between the role of child individual, parental and environmental risk factors. These results highlight the importance of considering developmental factors when assessing and targeting risk for anxiety.
ContributorsZerr, Argero (Author) / Pina, Armando A (Thesis advisor) / Bradley, Robert H (Committee member) / Doane, Leah D (Committee member) / Varela, Roberto E (Committee member) / Arizona State University (Publisher)
Created2012
Description
We used sex, observed parenting quality at 18 months, and three variants of the catechol-O-methyltransferase gene (Val[superscript 158]Met [rs4680], intron1 [rs737865], and 3′-untranslated region [rs165599]) to predict mothers' reports of inhibitory and attentional control (assessed at 42, 54, 72, and 84 months) and internalizing symptoms (assessed at 24, 30, 42, 48, and 54 months) in a sample of 146 children (79 male). Although the pattern for all three variants was very similar, Val[superscript 158]Met explained more variance in both outcomes than did intron1, the 3′-untranslated region, or a haplotype that combined all three catechol-O-methyltransferase variants. In separate models, there were significant three-way interactions among each of the variants, parenting, and sex, predicting the intercepts of inhibitory control and internalizing symptoms. Results suggested that Val[superscript 158]Met indexes plasticity, although this effect was moderated by sex. Parenting was positively associated with inhibitory control for methionine–methionine boys and for valine–valine/valine–methionine girls, and was negatively associated with internalizing symptoms for methionine–methionine boys. Using the “regions of significance” technique, genetic differences in inhibitory control were found for children exposed to high-quality parenting, whereas genetic differences in internalizing were found for children exposed to low-quality parenting. These findings provide evidence in support of testing for differential susceptibility across multiple outcomes.
ContributorsSulik, Michael (Author) / Eisenberg, Nancy (Author) / Spinrad, Tracy (Author) / Lemery, Kathryn (Author) / Swann, Gregory (Author) / Silva, Kassondra (Author) / Reiser, Mark (Author) / Stover, Daryn (Author) / Verrelli, Brian (Author) / College of Liberal Arts and Sciences (Contributor) / Department of Psychology (Contributor) / Sanford School of Social and Family Dynamics (Contributor) / School of Mathematical and Statistical Sciences (Contributor)
Created2015-08-01
Description
Children's chronic pain has many contributing factors, including family environment, genetics, and parenting. Still, pediatric chronic pain remains understudied, and little research has been conducted on predictors of child pain onset. This study aims to elucidate some of these factors by examining the role of parenting style and parental pain in children's chronic pain experience. The study answered the following questions: 1) Is child chronic pain heritable?; 2) Do parenting styles and/or parental pain predict child pain?; and 3) Is parenting style the mediating variable in the relation between parent pain and child pain? A twin study design was employed to account for both genetic and environmental influences in pain. Primary and secondary caregivers completed pain questionnaires regarding their own and their children's pain. The caregivers also completed questionnaires regarding their own parenting styles. Observer ratings were used as additional measures of primary caregiver parenting. Results indicated that child pain is heritable and that parental pain was significantly related to child pain. However, parenting style did not predict child pain and was not a mediator in the relationship between parental pain and child pain. Further research on other parenting factors or predictors of pain may lead to prevention of pediatric chronic pain or more effective management of child pain symptoms.
ContributorsPatel, Maya (Author) / Davis, Mary (Thesis director) / Lemery, Kathryn (Thesis director) / Department of Psychology (Contributor) / Barrett, The Honors College (Contributor)
Created2018-05
Description
Adolescent mental health problems are predicative of future problems such as depression, anxiety, ADHD, compulsive disorder, and substance use. Previous studies show that in emerging adulthood, the high prevalence and associated burdens of psychopathology increase vulnerability to disorders. These diagnoses are less common but are more severe and chronic (Tanner et al., 2009). The causes of these disorders are still being explored with recent studies showing that these mental health problems are genetically influenced. This makes understanding which gene that corresponds to what biological system is important in determining mental health. From recent studies, genes that code for calcium channels are good candidates for mental health problems. These voltage-gated channels are important mediators for physiological functions in the central nervous system and their activation provides unique responses within the brain. In a previous study, it supports the association of polymorphisms in calcium and potassium channels with the genetic risk for bipolar disorders and other mental illness (Imbrici et al., 2013). The purpose of the study was to examine if calcium channel genes influence childhood psychiatric symptoms. The first goal of this study was to form a polygenic risk score representing genetic influence on calcium channels. The second goal was to use this risk score in genetic association analyses to understand genetic risk for childhood psychopathology. Overall, the study did accomplish the goal as a polygenic risk score was created and was used to examine genetic association with child psychopathology. Based on the results, the polygenic risk score was not correlated with either parent or child- reported symptoms; however, results did show that disorders were related to each other and differed by race.
ContributorsTang, Derek (Author) / Lemery, Kathryn (Thesis director) / Gipson-Reichardt, Cassandra (Committee member) / Elam, Kit (Committee member) / School of Life Sciences (Contributor) / Barrett, The Honors College (Contributor)
Created2018-05
Description
The transition to college is a time of increased opportunity and stress that spans across multiple domains (e.g., social life, academic workload, finances). Adolescents who encounter significant stress during the transition to college may be vulnerable to adverse outcomes, due to a “wear and tear” of physiological systems, including the hypothalamic-pituitary-adrenal (HPA) axis. Latino students may be particularly at-risk for heightened stress exposure, as minority youth often experience both minority-specific stressors and general life stress. Despite this, the majority of research on Latino students is limited to the examination of singular forms of stress, and little is known regarding the cumulative impact of multiple forms of stress on Latino students’ HPA axis functioning. The present study employed a “multi-risk model approach” to examine the additive, common, and cumulative effects of multiple types of stress (general, academic, social, financial, bicultural, discrimination) on HPA axis functioning in Latino college students (N = 209; 64.4% female; Mage = 18.95). Results from three-level growth curve models indicated that, in the additive model, no stressors were associated with the CAR, but general stress was associated with a flatter diurnal cortisol slope (DCS) and bicultural stress was linked with a steeper DCS. In the common model, the college stress latent factor was related to a reduced cortisol awakening response (CAR), but not the DCS. In the cumulative model, cumulative risk was linked with a lower CAR, but not the DCS. These findings highlight the physiological correlates of various stressors experienced by Latino college students.
ContributorsSasser, Jeri (Author) / Doane, Leah D (Thesis advisor) / Su, Jinni (Committee member) / Grimm, Kevin J (Committee member) / Arizona State University (Publisher)
Created2022
Description
Various physical and psychological forms of development take place during the transition from childhood to adolescence, including the onset of puberty, delayed sleep-wake behavior patterns, and increases in internalizing symptomatology. Theory suggests that pubertal processes influence the onset of internalizing symptoms, and this association may differ between boys and girls. The contextual amplification hypothesis suggests that pubertal development interacts with contextual or dispositional factors to impact risk for psychopathology. Family stress and peer stress are two critical factors during early adolescence that have potential moderating effects on the association between pubertal development and internalizing symptoms. In line with the biopsychosocial framework, the current study examined various psychosocial (e.g., family stress, peer stress) and biological (e.g., sleep, genetics) factors that may have effects on the puberty-internalizing relation. Participants were a racially/ethnically and socioeconomically diverse sample of twins who were part of an ongoing longitudinal study (N=818 children; Arizona Twin Project; Lemery-Chalfant et al. 2019). The current study examined the association between puberty and internalizing symptoms, with stress (i.e., family, peer) as potential moderators, and sleep indicators (i.e., objective and self-reported sleep) as potential mediators. Behavior genetic analyses explored the moderated heritability of family stress on internalizing symptoms. Findings revealed that greater pubertal development predicted higher internalizing symptoms in boys, but not girls. For girls, peer stress interacted with pubertal development to predict internalizing symptoms, but simple slopes were not significant. Sleep indicators were not significant mediators between pubertal development and internalizing symptoms for boys or girls. Univariate twin models revealed environmental influences on internalizing symptoms, and both genetic and environmental influences on peer stress. Family stress did not significantly moderate the genetic and environmental influences of internalizing symptoms.
ContributorsLecarie, Emma (Author) / Doane, Leah D (Thesis advisor) / Lemery-Chalfant, Kathryn (Committee member) / Gewirtz, Abigail H (Committee member) / Grimm, Kevin J (Committee member) / Arizona State University (Publisher)
Created2024
Description
This dissertation combines three first-author manuscripts that focused broadly on the study of adolescent sleep within a family context (Sasser et al., 2021; Sasser & Oshri, 2023; Sasser et al., 2023). First, Chapter 1 introduces the theoretical background and empirical research that grounded the research questions and hypotheses explored across the studies. The first study (Chapter 2) examined the influence of family connection on actigraphy-measured sleep among Latinx late adolescents and explored family dynamics and cultural values as potential moderators. The second study (Chapter 3) investigated daily and average concordance between parent and youth actigraphy-measured sleep and how this varied as a function of family context (e.g., parenting, family functioning). The third study (Chapter 4) examined concordance in actigraphy sleep among parent-youth and sibling dyads and explored how relations differed across zygosity type and sleeping arrangements. The dissertation concludes with an immersive discussion (Chapter 5) that summarizes the key differences, similarities, and takeaways across studies and highlights future directions and implications for developmental science, public policy, and clinical interventions. Collectively, this dissertation contributes to the understanding of youth and adolescent sleep within a family context by identifying proximal (e.g., daily interactions with parents/siblings) and broader family-level factors (e.g., dynamics, culture) that may help promote more healthful sleep among both adolescents and their family members.
ContributorsSasser, Jeri (Author) / Doane, Leah D (Thesis advisor) / Su, Jinni (Committee member) / Grimm, Kevin J (Committee member) / Lemery-Chalfant, Kathryn (Committee member) / Arizona State University (Publisher)
Created2023