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- Genre: Masters Thesis
Description
Research suggests that early family relationships have critical influences on later physical and psychological health, but most studies have focused on the influence of mothers ignoring the unique impacts of fathers. One mechanism by which families may transmit risk is by repeated activation of the hypothalamic-pituitary-adrenal (HPA) axis in the short-term that leads to adult neurobiological dysregulaton, evident in hyper- or hypo-cortisol levels. Using 218 father-child dyads from the Parent and Youth Study (PAYS), the current study investigated whether father involvement in adolescence predicted youth cortisol AUCg and reactivity to a stress task in young adulthood, and whether this relation was mediated by youth perceptions of mattering to their fathers in adolescence. Results revealed that higher father-reported father involvement predicted lower cortisol AUCg in youth when mattering was included in the model, although father involvement was not a statistically significant predictor of AUCg or cortisol reactivity when mattering was not included. Additionally, children who reported higher father involvement also reported higher feelings of mattering, but this association was only statistically significant for girls and European American youth. Youth feelings of mattering did not predict their cortisol reactivity or AUCg in young adulthood. Results suggest that future research should include fathers when investigating the effects of family relationships on youth psychophysiological development.
ContributorsHanna, Mariam A (Author) / Luecken, Linda (Thesis advisor) / Wolchik, Sharlene (Committee member) / Fabricius, William (Committee member) / Arizona State University (Publisher)
Created2015
Description
Childhood obesity is associated with many well established health risks as well as high annual public health costs. Because of this, the childhood obesity literature has highlighted the need to detect at-risk groups in order to implement targeted preventions. Emotional eating has been identified as an unhealthy behavior and a risk factor for overweight status among children though very little is known about what predisposes children to emotionally eat. Stress has often been found to elicit emotional eating but most studies looking at this relationship have relied on self-reports in adult and clinical samples. Thus, the current study seeks to investigate the relationship between stress reactivity (measured using heart rate variability) and emotional eating in a sample of 247 children between the ages of 4-6. Furthermore, levels of control may moderate the relationship between stress reactivity (HRV) and emotional eating. Linear regression analysis was used to explore these relationships. It was expected that higher levels of reactivity to stress would predict an increased likelihood of emotional eating. This association was expected to be attenuated among children with higher levels of inhibitory control and attentional focusing as well as lower levels of impulsivity. However, the hypothesized findings were not supported by the data. Despite these null findings, and in light of several limitations, it is still hypothesized that emotional eating involves physiological and impulsivity/effortful control processes. Implications of future research are discussed.
ContributorsOhrt, Tara K (Author) / Perez La Mar, Marisol (Thesis advisor) / Luecken, Linda (Committee member) / Lemery, Kathryn (Committee member) / Arizona State University (Publisher)
Created2016
Description
The theory of biological sensitivity to context (BSC; Boyce & Ellis, 2005) posits that specific biological characteristics, such as vagal tone, may confer risk for physical and mental health outcomes for some children but promote health for others. High levels of resting respiratory sinus arrhythmia (RSA), an index of vagal tone, may confer susceptibility to the effects of the caregiving environment on child development. Consistent with BSC, I expected that, relative to infants with lower RSA, infants with higher RSA would demonstrate fewer behavior problems if their mothers reported fewer postpartum depressive symptoms, but more behavior problems if their mothers reported more postpartum depressive symptoms. I also evaluated whether observed child social engagement with their mothers mediated children's biological sensitivity to the effects of postpartum depressive symptoms on behavior problems in early childhood. I evaluated a mediated moderation model among a sample of 322 low-income Mexican American mother-infant dyads. As expected, the RSA x maternal depressive symptoms interaction, controlling for covariates, was a significant predictor of internalizing, externalizing and total behavior problems, and high vagal tone conferred susceptibility for externalizing behavior problems. Contrary to my hypothesis, children with low RSA may be more susceptible to the effects of maternal postpartum depressive symptoms on children's internalizing and total behavior problems, and child social engagement did not account for these effects. Among infants in economically disadvantaged families, lower RSA and fewer maternal depressive symptoms may promote resilience, and more research is needed to understand behavioral mediators of biological sensitivity.
ContributorsSomers, Jennifer (Author) / Luecken, Linda (Thesis advisor) / Crnic, Keith (Committee member) / Spinrad, Tracy (Committee member) / Arizona State University (Publisher)
Created2017
Description
Dysregulated cortisol has been linked to a variety of adverse physical and psychological consequences. Stressors in the childhood family environment can influence cortisol activity throughout development. For example, research has shown that both infants and children of depressed mothers exhibit altered levels of cortisol compared to infants and children of non-depressed mothers. It is unclear, however, whether exposure to maternal depression in childhood and adolescence is related to cortisol activity at later stages of development. The current study examined the longitudinal relation between maternal depressive symptoms during late childhood (9-12 years old) and adolescence (15-19 years old) and cortisol activity in offspring in young adulthood (24- 28 years old) in a sample of 40 young adults and their mothers. Maternal depressive symptoms were prospectively assessed at four time points across the 15 year study. Cortisol samples were collected from young adult offspring at the final time point. Findings revealed that higher levels of maternal depressive symptoms during late childhood were associated with lower total cortisol output in young adulthood. Results suggest that attenuated cortisol levels, which put these young adults at risk for a variety of stress-related physical and psychological illnesses, may be a long-term consequence of exposure to maternal depression,. Depressive symptoms in mothers during their child's adolescence, however, did not relate to cortisol output. These findings suggest a sensitive period in late childhood during which the development of HPA activity may be susceptible to the environmental stressor of maternal depression.
ContributorsMahrer, Nicole Eva (Author) / Wolchik, Sharlene (Thesis advisor) / Luecken, Linda (Thesis advisor) / Tein, Jenn-Yun (Committee member) / Arizona State University (Publisher)
Created2011
Examination of Shame as a Mediator of the Relationship Between Food Insecurity and Disordered Eating
Description
Background: An emerging literature has found associations between food insecurity and disordered eating behaviors. This study was two-fold. First, to replicate the existing literature that food insecurity is associated with disordered eating symptoms including loss of control over eating. Second, to expand the existing literature by examining stigma control theory, which purports that experiences related to food insecurity potentially induce stigma-related shame, with disordered eating behaviors used to cope with the shame. Further, to explore if emotion coping strategies moderate associations between shame and disordered eating. Method: This is a secondary analysis of a cross-sectional study of 582 adults with food insecurity. Participants completed a 20-minute online survey on food insecurity, shame related to food insecurity, coping strategies, and disordered eating behaviors. Analyses: Hierarchical regressions were computed where food insecurity, shame, and emotion focused coping were entered as predictor variables, followed by their interaction terms, and with disordered eating behaviors entered as outcome variables. Results: Regressions suggest that a) internalized shame partially mediated the relationship between food insecurity and global disordered eating, b) internalized shame did not mediate the relationship between food insecurity and loss of control, c) emotion focused coping did not moderate any relationship. Discussion: Internalized shame may be one mechanism in which disordered symptoms arise in food insecure populations, however emotion focused coping does not have any effect on this relationship. Results indicate that coping strategies alone may not reduce eating disorder symptoms, and internalized shame may be an important predictor of disordered eating in food insecure populations.
ContributorsGomez, Francesca (Author) / Perez, Marisol (Thesis advisor) / Berkel, Cady (Committee member) / Luecken, Linda (Committee member) / Arizona State University (Publisher)
Created2022
Description
Internalizing symptoms are prevalent among adolescents, especially among Latinos, and can have negative consequences on health and development. Understanding the risk and protective factors leading to internalizing difficulties among Latino youth is critical. The current study sought to assess the effects of family risk and peer social rejection in the seventh grade on internalizing symptoms in the tenth grade, and the potential buffering effects of social support from family and from friends, among a sample of 749 Mexican American youth. Structural equation modeling was used to examine pathways from seventh grade family risk and peer social rejection to internalizing symptoms in the tenth grade. Perceived social support from family and perceived social support from friends were tested as moderators of these relations. Gender differences in these pathways were also assessed. Results showed that family risk did not predict tenth grade internalizing symptoms, but that peer social rejection predicted increased internalizing symptoms for girls. Furthermore, buffering effects were not confirmed; rather social support from both friends and family had no effect on the relation between family risk and internalizing symptoms, and high levels of social support from both sources amplified the effect of peer social rejection on internalizing symptoms. Secondary analyses suggested that at low levels of social support from both sources, peer social rejection predicted decreased internalizing symptoms for males. Limitations and implications for prevention and future research are discussed.
ContributorsJenchura, Emily C (Author) / Gonzales, Nancy (Thesis advisor) / Tein, Jenn-Yun (Committee member) / Luecken, Linda (Committee member) / Arizona State University (Publisher)
Created2015
Description
Ecological systems theory argues that multiple nested systems impact child development. This study used a moderated mediation pathway to examine whether presence of a grocery store, number of fast-food restaurants, outdoor play space, and outdoor play safety affected children’s blood pressure and BMI through variation in healthy family habits. Maternal perceived neighborhood social and cultural cohesion was examined as a moderator of the mediated effect. Data was collected from 214 mother–child dyads via biological measurement, maternal-report surveys, and geocoding of children’s neighborhoods using Google Earth. Zero-order correlations showed that higher number of fast-food restaurants in a child’s neighborhood was correlated with less engagement in healthy family habits and lower child BMI z-score. In all models, higher neighborhood social and cultural cohesion was associated with more engagement in healthy family habits. No statistically significant mediated effects or moderation of the mediated effects were found. Future directions may aim to identify which objective neighborhood environment indicators influence child health and what are potential variables mediating the relation.
ContributorsHernandez, Juan Carlos (Author) / Perez, Marisol (Thesis advisor) / Luecken, Linda (Committee member) / Anderson, Samantha (Committee member) / White, Rebecca (Committee member) / Arizona State University (Publisher)
Created2020