Filtering by
- Creators: Honeycutt, Claire
- Creators: School of Life Sciences
- Creators: Niebuhr, Robert
- Creators: Ossanna, Meilin Ryan
- Member of: Barrett, The Honors College Thesis/Creative Project Collection
This paper will discuss what psychological operations (PSYOP) and psychological warfare (PSYWAR) are, emphasizing the role they played in the Korean War on soldiers and POWs. It includes two dynamic, engaging personal narratives adding relevance and intrigue to the topic examined. Two types of psychological operations will be identified and discussed, the leaflet and radio broadcast. Three methods of psychological warfare in the form of bacteriological warfare allegations, brainwashing, and sleep deprivation are analyzed and the effects of these methods during wartime. All participating countries during the Korean war either used psychological operations and warfare on their enemies, or had it used on them. The key takeaway was increased understanding that PSYOP and PSYWAR are founded on truthfulness rather than falsehoods. The paper concludes with the results of the war on the players involved and the legacy of psychological operations, encompassing how it applies to our technologically advanced world today.
Along with aging, sleep deprivation is correlated with learning deficits. Research has shown that a lack of sleep negatively impacts motor skill learning and consolidation. Since there is a link between sleep and learning, as well as learning and the reticulospinal system, these observations raise the question: does sleep deprivation underlie reticulospinal delays? We hypothesized that sleep deprivation was correlated to a slower startle response, indicating a delayed reticulospinal system. Our objectives were to observe the impact of sleep deprivation on 1) the startle response (characterized by muscle onset latency and percentage of startle responses elicited) and 2) functional performance (to determine whether subjects were sufficiently sleep deprived).
21 young adults participated in two experimental sessions: one control session (8-10 hour time in bed opportunity for at least 3 nights prior) and one sleep deprivation session (0 hour time in bed opportunity for one night prior). The same protocol was conducted during each session. First, subjects were randomly exposed to 15 loud, startling acoustic stimuli of 120 dB. Electromyography (EMG) data measured muscle activity from the left and right sternocleidomastoid (LSCM and RSCM), biceps brachii, and triceps brachii. To assess functional performance, cognitive, balance, and motor tests were also administered. The EMG data were analyzed in MATLAB. A generalized linear mixed model was performed on LSCM and RSCM onset latencies. Paired t-tests were performed on the percentage of startle responses elicited and functional performance metrics. A p-value of less than 0.05 indicated significance.
Thirteen out of 21 participants displayed at least one startle response during their control and sleep deprived sessions and were further analyzed. No differences were found in onset latency (RSCM: control = 75.87 ± 21.94ms, sleep deprived = 82.06 ± 27.47ms; LSCM: control = 79.53 ± 17.85ms, sleep deprived = 78.48 ± 20.75ms) and percentage of startle responses elicited (control = 84.10 ± 15.53%; sleep deprived = 83.59 ± 18.58%) between the two sessions. However, significant differences were observed in reaction time, TUG with Dual time, and average balance time with the right leg up. Our data did not support our hypothesis; no significant differences were seen between subjects’ startle responses during the control and sleep deprived sessions. However, sleep deprivation was indicated with declines were observed in functional performance. Therefore, we concluded that sleep deprivation may not affect the startle response and underlie delays in the reticulospinal system.