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Presented is a study on the chemotaxis reaction process and its relation with flow topology. The effect of coherent structures in turbulent flows is characterized by studying nutrient uptake and the advantage that is received from motile bacteria over other non-motile bacteria. Variability is found to be dependent on the

Presented is a study on the chemotaxis reaction process and its relation with flow topology. The effect of coherent structures in turbulent flows is characterized by studying nutrient uptake and the advantage that is received from motile bacteria over other non-motile bacteria. Variability is found to be dependent on the initial location of scalar impurity and can be tied to Lagrangian coherent structures through recent advances in the identification of finite-time transport barriers. Advantage is relatively small for initial nutrient found within high stretching regions of the flow, and nutrient within elliptic structures provide the greatest advantage for motile species. How the flow field and the relevant flow topology lead to such a relation is analyzed.
ContributorsJones, Kimberly (Author) / Tang, Wenbo (Thesis advisor) / Kang, Yun (Committee member) / Jones, Donald (Committee member) / Arizona State University (Publisher)
Created2015
Description
A continuously and stably stratified fluid contained in a square cavity subjected to harmonic body forcing is studied numerically by solving the Navier-Stokes equations under the Boussinesq approximation. Complex dynamics are observed near the onset of instability of the basic state, which is a flow configuration that is always an

A continuously and stably stratified fluid contained in a square cavity subjected to harmonic body forcing is studied numerically by solving the Navier-Stokes equations under the Boussinesq approximation. Complex dynamics are observed near the onset of instability of the basic state, which is a flow configuration that is always an exact analytical solution of the governing equations. The instability of the basic state to perturbations is first studied with linear stability analysis (Floquet analysis), revealing a multitude of intersecting synchronous and subharmonic resonance tongues in parameter space. A modal reduction method for determining the locus of basic state instability is also shown, greatly simplifying the computational overhead normally required by a Floquet study. Then, a study of the nonlinear governing equations determines the criticality of the basic state's instability, and ultimately characterizes the dynamics of the lowest order spatial mode by the three discovered codimension-two bifurcation points within the resonance tongue. The rich dynamics include a homoclinic doubling cascade that resembles the logistic map and a multitude of gluing bifurcations.

The numerical techniques and methodologies are first demonstrated on a homogeneous fluid contained within a three-dimensional lid-driven cavity. The edge state technique and linear stability analysis through Arnoldi iteration are used to resolve the complex dynamics of the canonical shear-driven benchmark problem. The techniques here lead to a dynamical description of an instability mechanism, and the work serves as a basis for the remainder of the dissertation.
ContributorsYalim, Jason (Author) / Welfert, Bruno D. (Thesis advisor) / Lopez, Juan M. (Thesis advisor) / Jones, Donald (Committee member) / Tang, Wenbo (Committee member) / Platte, Rodrigo (Committee member) / Arizona State University (Publisher)
Created2019
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Description
Using a simple $SI$ infection model, I uncover the

overall dynamics of the system and how they depend on the incidence

function. I consider both an epidemic and endemic perspective of the

model, but in both cases, three classes of incidence

functions are identified.

In the epidemic form,

power incidences, where the infective portion $I^p$

Using a simple $SI$ infection model, I uncover the

overall dynamics of the system and how they depend on the incidence

function. I consider both an epidemic and endemic perspective of the

model, but in both cases, three classes of incidence

functions are identified.

In the epidemic form,

power incidences, where the infective portion $I^p$ has $p\in(0,1)$,

cause unconditional host extinction,

homogeneous incidences have host extinction for certain parameter constellations and

host survival for others, and upper density-dependent incidences

never cause host extinction. The case of non-extinction in upper

density-dependent

incidences extends to the case where a latent period is included.

Using data from experiments with rhanavirus and salamanders,

maximum likelihood estimates are applied to the data.

With these estimates,

I generate the corrected Akaike information criteria, which

reward a low likelihood and punish the use of more parameters.

This generates the Akaike weight, which is used to fit

parameters to the data, and determine which incidence functions

fit the data the best.

From an endemic perspective, I observe

that power incidences cause initial condition dependent host extinction for

some parameter constellations and global stability for others,

homogeneous incidences have host extinction for certain parameter constellations and

host survival for others, and upper density-dependent incidences

never cause host extinction.

The dynamics when the incidence function is homogeneous are deeply explored.

I expand the endemic considerations in the homogeneous case

by adding a predator into the model.

Using persistence theory, I show the conditions for the persistence of each of the

predator, prey, and parasite species. Potential dynamics of the system include parasite mediated

persistence of the predator, survival of the ecosystem at high initial predator levels and

ecosystem collapse at low initial predator levels, persistence of all three species, and much more.
ContributorsFarrell, Alexander E. (Author) / Thieme, Horst R (Thesis advisor) / Smith, Hal (Committee member) / Kuang, Yang (Committee member) / Tang, Wenbo (Committee member) / Collins, James (Committee member) / Arizona State University (Publisher)
Created2017