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ContributorsPerkins, Caitlin (Author) / Jacobs, Bertram (Thesis director) / Gile, Gillian (Committee member) / Barrett, The Honors College (Contributor) / School of Life Sciences (Contributor)
Created2023-05
ContributorsPerkins, Caitlin (Author) / Jacobs, Bertram (Thesis director) / Gile, Gillian (Committee member) / Barrett, The Honors College (Contributor) / School of Life Sciences (Contributor)
Created2023-05
Description

In completing this thesis project, I attempted to hypothesize the trigger in my own personal diagnosis of type 1 diabetes through literature research as well as further research on viruses and their contribution to autoimmune disorders. I had previously hypothesized that, based on my own family life, type 1 diabetes

In completing this thesis project, I attempted to hypothesize the trigger in my own personal diagnosis of type 1 diabetes through literature research as well as further research on viruses and their contribution to autoimmune disorders. I had previously hypothesized that, based on my own family life, type 1 diabetes could possibly be a non-heritable disease despite its consistent inheritance pattern discovered by researchers; however, the research presented in this thesis project rejects this idea and supports the theory that I may have been previously susceptible to this disorder and would have developed type 1 diabetes naturally. There were multiple viruses discovered during the literature research conducted that could possibly have been triggers in the acceleration of my disease. The major link between enteroviruses and autoimmune disorders was discovered, as well as influenza A and SARS-COV-2 and this is explained further in this project.

ContributorsPerkins, Caitlin (Author) / Jacobs, Bertram (Thesis director) / Gile, Gillian (Committee member) / Barrett, The Honors College (Contributor) / School of Life Sciences (Contributor)
Created2023-05