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- All Subjects: parenting
- Creators: Chassin, Laurie
- Creators: Lemery, Kathryn
Analyses were performed in a path analysis framework. To test these research questions, the current study employed two polygenic risk scores. The first, a theory-based score, was formed using single-nucleotide polymorphisms (SNPs) from receptor systems implicated in the amplification of positive effects in the presence of new/exciting stimuli and/or pleasure derived from using substances. The second, an empirically-based score, was formed using a data-driven approach that explained a large amount of variance in SUDs. Together, these scores allowed the present study to test explanations for the relations among parent AUD, parental knowledge, peer substance use, and SUDs.
Results of the current study found that having parents with less knowledge or an AUD conferred greater risk for SUDs, but only for those at higher genetic risk for behavioral undercontrol. The current study replicated research findings suggesting that peer substance use mediated the effect of parental AUD on SUD. However, it adds to this literature by suggesting that some mechanism other than increased behavioral undercontrol explains relations among parental AUD, peer substance use, and emerging adult SUD. Taken together, these findings indicate that children of parents with AUDs comprise a particularly risky group, although likelihood of SUD within this group is not uniform. These findings also suggest that some of the most important environmental risk factors for SUDs exert effects that vary across level of genetic propensity.
This cross-sectional study examined whether the temperament dimensions of negative emotionality, positive emotionality, and impulsivity moderated the relation between interparental conflict and children’s internalizing and externalizing problems. The sample consisted of 355 divorced mothers and their children (9-12 years old) who participated in a randomized controlled trial of a preventive parenting intervention for divorcing families. Children provided reports of their experiences of interparental conflict and internalizing and externalizing problems; mothers provided reports of children’s temperament and internalizing and externalizing problems. The relations were examined separately for child report and mother report of outcomes using multiple regression analyses. Results found no support for the interactive effect of interparental conflict and temperament dimensions on children’s internalizing or externalizing problems. Consistent with an additive model of their effects, interparental conflict and temperament dimensions were directly and independently related to the outcomes. There was a significant, positive effect of interparental conflict and negative emotionality on children’s internalizing and externalizing problems. Positive emotionality was significantly, negatively related to internalizing and externalizing problems. Impulsivity was significantly, positively related to externalizing problems only. The patterns of results varied somewhat across mother and child report of interparental conflict on externalizing problems and positive emotionality on internalizing problems. The results of this study are consistent with the previous research on the significant main effects of interparental conflict and temperament dimensions on children’s internalizing and externalizing problems. These findings suggest that children’s environment and intrapersonal characteristics, represented by children’s experiences of interparental conflict and temperament, both uniquely contribute to children’s post-divorce internalizing and externalizing problems.
Adolescence is an important period of scaffolding for educational attainment, which is among the strongest predictors of outcomes in multiple domains. Parents who encourage academic success and promote self-regulation may enhance their offspring’s educational attainment. However, parents with externalizing disorders present a complex constellation of risk factors, including low educational attainment and poor parenting, and are more likely to have children with high levels of disinhibition. Previous research has identified low parental education, poor parenting and adolescent impulsivity as threats to educational attainment, but has not examined risk factors for discrepancies in educational attainment among siblings of the same family. Furthermore, studies have not examined the between- and within-family mechanisms that may explain why adolescents with externalizing parents have low educational attainment. The current study addressed these gaps by testing between- and within-family predictors of educational attainment using data from a longitudinal, multigenerational study that oversampled families at risk for alcohol use disorder. The sample consisted of 555 biological siblings within 240 families. We tested whether parental externalizing predicted lower educational attainment through parents’ own lower education, parents’ differential treatment of offspring, and impulsivity. Results suggested that between families, parents with externalizing disorders had lower educational attainment and more impulsive offspring, but did not exhibit increased differential parenting. Within families, siblings with greater impulsivity had lower educational attainment, whereas receiving more preferential maternal treatment than one’s siblings predicted higher educational attainment. Low parental educational attainment mediated the relation between parental externalizing disorders and low offspring educational attainment.
Participants (N = 236 in models that included multiple G2 siblings; N = 110 in models without siblings) were drawn from a larger sample of at-risk (i.e., alcoholic) and comparison families followed longitudinally for over 30 years and across three generations. Four mediation models were estimated to examine effects of two separate G1-G2 family disruption components (deviance-related and health-related disruption) on parenting of G3, mediated by family conflict, family disorganization, and G2 dysregulation. Results indicated that health-related disruption impairs consistency of parenting provided to G3 offspring through conflict in the G1-G2 family. A direct effect of health-related disruption was also seen on parental monitoring. There were no direct or mediated effects of deviance-related disruption on parenting. Implications and future directions will be discussed.
Prior research has established a relation between parenting behaviors and symptoms of child psychopathology, and this association may be influenced by both genetic and environmental factors. Gene-environment correlation, or the influence of a child’s genes on the environment they receive, represents one possible mechanism through which genes and environment combine to influence child outcomes. This study examined evocative gene-environment correlation in the relation between parenting and symptoms of child psychopathology in a sample of 676 twins (51.5% female, 58.5% Caucasian, 23.7% Hispanic/Latinx, primarily middle class, MAge=8.43, SD=.62) recruited from Arizona birth records. Using univariate ACE twin biometric models, genetic influences were found to moderately contribute to internalizing symptoms (A=.47, C=.25, E=.28), while externalizing (A=.86, E=.14) and ADHD (A=.84, E=.16) symptoms were found to be highly heritable. The genetic influences for positive (C=.54, E=.46) and negative (C=.44, E=.56) parenting were smaller and found to be nonsignificant. The correlations between parenting and types of psychopathology were examined and bivariate Cholesky decompositions were conducted for statistically significant correlations. Negative parenting was moderately positively correlated with externalizing and ADHD symptoms; the relation between externalizing symptoms and negative parenting was found to be due to shared genetics, whereas the relation between negative parenting and ADHD symptoms was due to the shared environment. The mixed results regarding the role of gene environment correlation in relations between parenting and child psychopathology indicate that further research on the mechanisms of this relation is needed.