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- All Subjects: Cancer
- Creators: Department of Psychology
In study 1, I investigated how parents’ endorsements of values associated with both mainstream and heritage cultures relate to adolescents’ bicultural competence. Longitudinal growth model analyses revealed that parents’ endorsements of mainstream and heritage values simultaneously work to influence adolescents’ bicultural competence. By examining the effect of multiple and often competing familial contextual influences on adolescent bicultural competence development, this work provides insights on intergenerational cultural transmission and advances scholarship on the culturally bounded nature of human development.
In study 2, I offer a substantial extension to decades of family stress model research focused on how family environmental stressors may compromise parenting behaviors and youth development by testing a culturally informed family stress model. My model (a) incorporates family cultural and ecological stressors, (b) focuses on culturally salient parenting practices aimed to teach youth about the heritage culture (i.e., ethnic socialization), and (c) examines bicultural competence as a developmental outcome. Findings suggest that parents’ high exposure to ecological stressors do not compromise parental ethnic socialization or adolescent bicultural competence development. On the other hand, mothers’ exposures to enculturative stressors can disrupt maternal ethnic socialization, and in turn, undermine adolescents’ bicultural competence. By examining the influence of multiple family environmental stressors on culturally salient parenting practices, and their implications for adolescent bicultural competence development, this work provides insights on ethnic-racial minority and immigrant families’ adapting cultures and advances scholarship on the family stress model.
The goal of this project was to design and create a genetic construct that would allow for <br/>tumor growth to be induced in the center of the wing imaginal disc of Drosophila larvae, the <br/>R85E08 domain, using a heat shock. The resulting transgene would be combined with other <br/>transgenes in a single fly that would allow for simultaneous expression of the oncogene and, in <br/>the surrounding cells, other genes of interest. This system would help establish Drosophila as a <br/>more versatile and reliable model organism for cancer research. Furthermore, pilot studies were <br/>performed, using elements of the final proposed system, to determine if tumor growth is possible <br/>in the center of the disc, which oncogene produces the best results, and if oncogene expression <br/>induced later in development causes tumor growth. Three different candidate genes were <br/>investigated: RasV12, PvrACT, and Avli.