Matching Items (2)
Description
Sleep and weight health during childhood can be the essential building blocks that influence later physical and psychological health. Researchers note how neighborhood effects on health may be mediated or moderated by parenting or family-related factors. This dissertation expanded on the efforts through an examination of contextual predictors of children’s health using the Arizona Twin Project dataset (N = 933; 51.7% female; 56.5% White; 25.6% Latino). The family stress model (FSM), contextual relevance model (CRM), and the integrated model were used to test the relations between neighborhood and family factors on children sleep and weight health. Parent perceptions of neighborhood danger and childhood opportunities index were tested using multilevel- mediation, moderation, and moderated mediation models to address some of the limitations in previous reviews. The FSM was partially supported: parental stress and strain mediated the association between perceived neighborhood danger with sleep efficiency. However, the FSM was not fully supported as parental stress and disrupted parenting did not serially mediate the associations between neighborhood conditions and sleep efficiency. Moderation analyses revealed that negative parenting exacerbates associations between perceived neighborhood danger and sleep duration, demonstrating amplified disadvantages processes of the CRM. Negative parenting also moderated the associations between perceived neighborhood danger and sleep efficiency; the relationship was beyond what was noted in the CRM. Greater neighborhood opportunity was more strongly related to greater body fat percentage in families with higher positive parenting, as compared to families with lower positive parenting. Familism significantly moderated the association between neighborhood contexts and parental stress and strain within a subsample (Latino descent only). Last, the exploratory multilevel moderated mediation models suggested that most associations did not vary by the subgroups explored (i.e., positive parenting, familism cultural value). These results join a growing initiative to explore the science of ecological context, culture, and family interplay.
ContributorsPark, HyeJung (Author) / Doane, Leah D. (Thesis advisor) / White, Rebecca M.B. (Committee member) / Lemery-Chalfant, Kathryn (Committee member) / Su, Jinni (Committee member) / Arizona State University (Publisher)
Created2022
Description
Pediatric chronic pain is pervasive and associated with myriad adverse consequences, yet due consideration has not been given to the mental health disturbances that often present alongside chronic pain and the etiological mechanisms that potentially underlie both. The current study examined the etiology underlying chronic pain and internalizing symptomology in middle childhood, considering both independent and co-occurring symptom presentations. Phenotypic parent-offspring associations across chronic pain and internalizing symptomology were also examined. Lastly, nuclear twin family models were tested to determine the extent to which genetic and environmental factors underlie parent-offspring transmission. The sample comprised 795 children (399 families; Mage= 9.7 years; SD = 0.92) and their parents drawn from the Arizona Twin Project. Results indicated that chronic pain was highly heritable (78%), whereas internalizing symptomology was modestly heritable (32%) and further subject to moderate shared environmental influence (50%). Moreover, 9% of the variance in chronic pain was explained by additive genetic factors shared with internalizing symptomology. Maternal chronic pain and internalizing symptomology were positively associated with both child chronic pain and internalizing symptomology. The association between maternal chronic pain and child chronic pain was more pronounced for girls than boys, whereas the association between maternal internalizing symptomology and child internalizing symptomology was more pronounced for boys than girls. Paternal chronic pain was not significantly associated with child chronic pain but was unexpectedly associated with lower child internalizing symptomology. The negative association between paternal chronic pain and child internalizing symptomology was more pronounced for boys than girls. Paternal internalizing symptomology was not significantly associated with child chronic pain but was positively associated with child internalizing symptomology. Lastly, the best fitting reduced nuclear twin family models for both chronic pain and internalizing symptomology retained additive genetic, sibling-specific shared environmental, and nonshared environmental parameters, where parent-offspring transmission was solely explained by shared genetics and sibling-specific shared environmental factors further accounted for co-twin resemblance. Results provide novel insight into common liabilities underlying chronic pain and internalizing symptomology in middle childhood, parent-offspring associations across chronic pain and internalizing symptomology, and the etiological mechanisms that explain symptom aggregation across generations.
ContributorsOro, Veronica (Author) / Lemery-Chalfant, Kathryn (Thesis advisor) / Chassin, Laurie (Committee member) / Davis, Mary (Committee member) / Su, Jinni (Committee member) / Arizona State University (Publisher)
Created2021