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- All Subjects: psychology
- Genre: Academic theses
- Genre: Masters Thesis
Description
The development of self-regulation is believed to play a crucial role in predicting later psychopathology and is believed to begin in early childhood. The early postpartum period is particularly important in laying the groundwork for later self-regulation as infants' dispositional traits interact with caregivers' co-regulatory behaviors to produce the earliest forms of self-regulation. Moreover, although emerging literature suggests that infants' exposure to maternal stress even before birth may be integral in determining children's self-regulatory capacities, the complex pathways that characterize these developmental processes remain unclear. The current study considers the complex, transactional processes in a high-risk, Mexican American sample. Data were collected from 305 Mexican American infants and their mothers during prenatal, 6- and 12-week home interviews. Mother self-reports of stress were obtained prenatally between 34-37 weeks gestation. Mother reports of infant temperamental negativity and surgency were obtained at 6-weeks as were observed global ratings of maternal sensitivity during a structured peek-a-boo task. Microcoded ratings of infants' engagement orienting and self-comforting behaviors were obtained during the 12-week peek-a-boo task. Study findings suggest that self-comforting and orienting behaviors help to modulate infants' experiences of distress, and also that prenatal stress influences infants' engagement in each of those regulatory behaviors, both directly by influence tendencies to engage in orienting behaviors and indirectly by programming higher levels of infant negativity and surgency, both of which may confer risk for later regulatory disadvantage. Advancing our understandings about the nature of these developmental pathways could have significant implications for targets of early intervention in this high-risk population.
ContributorsLin, Betty (Author) / Crnic, Keith A (Thesis advisor) / Lemery-Chalfant, Kathryn S (Committee member) / Mackinnon, David P (Committee member) / Arizona State University (Publisher)
Created2013
Description
Methods to test hypotheses of mediated effects in the pretest-posttest control group design are understudied in the behavioral sciences (MacKinnon, 2008). Because many studies aim to answer questions about mediating processes in the pretest-posttest control group design, there is a need to determine which model is most appropriate to test hypotheses about mediating processes and what happens to estimates of the mediated effect when model assumptions are violated in this design. The goal of this project was to outline estimator characteristics of four longitudinal mediation models and the cross-sectional mediation model. Models were compared on type 1 error rates, statistical power, accuracy of confidence interval coverage, and bias of parameter estimates. Four traditional longitudinal models and the cross-sectional model were assessed. The four longitudinal models were analysis of covariance (ANCOVA) using pretest scores as a covariate, path analysis, difference scores, and residualized change scores. A Monte Carlo simulation study was conducted to evaluate the different models across a wide range of sample sizes and effect sizes. All models performed well in terms of type 1 error rates and the ANCOVA and path analysis models performed best in terms of bias and empirical power. The difference score, residualized change score, and cross-sectional models all performed well given certain conditions held about the pretest measures. These conditions and future directions are discussed.
ContributorsValente, Matthew John (Author) / MacKinnon, David (Thesis advisor) / West, Stephen (Committee member) / Aiken, Leona (Committee member) / Enders, Craig (Committee member) / Arizona State University (Publisher)
Created2015
Description
Comorbidity is a significant problem for current classification systems of psychopathology (i.e., DSM-V). One issue is that the underlying mechanisms shared among comorbid disorders are poorly understood, especially potential psychosocial mechanisms such as family dynamics. The current study used latent class analysis to empirically classify patterns of psychopathology within a large community sample of late adolescents (age 18-19) based on their lifetime psychological adjustment measured using the World Health Organization Composite International Diagnostic Interview. Videotaped family interactions of adolescents (age 16-17) and their parents were micro and macro coded and the resulting family dynamics were compared across the three empirically defined groups of psychological adjustment which emerged from the latent class analysis: 1) an early onset, persistent antisocial behavior class; 2) an emotionally distressed and substance using class; and 3) a typically developing class. It was found that some directly observed family dynamics, including parental monitoring, dyadic positive engagement and coercive engagement discriminated among empirically derived classes. It was also found that particular tasks better discriminated among classes with regard to specific family dynamics (e.g., family activity task best discriminated among classes on dyadic positive engagement). Overall, findings suggest that novel methodologies like latent class analysis can be useful in attempting to map underlying transdiagnostic mechanisms onto the current diagnostic framework. The findings also highlight the importance of taking many variables into consideration when attempting to understand how family dynamics are associated with psychological adjustment.
ContributorsPanza, Kaitlyn E (Author) / Dishion, Thomas J (Thesis advisor) / Crnic, Keith A (Committee member) / Tein, Jenn-Yun (Committee member) / Arizona State University (Publisher)
Created2015
Description
Postpartum depression (PPD) is a significant public health concern affecting up to half a million U.S. women annually. Mexican-American women experience substantially higher rates of PPD, and represent an underserved population with significant health disparities that put these women and their infants at greater risk for substantial psychological and developmental difficulties. The current study utilized data on perceived stress, depression, maternal parenting behavior, and infant social-emotional and cognitive development from 214 Mexican-American mother-infant dyads. The first analysis approach utilized a latent intercept (LI) model to examine how overall mean levels and within-person deviations of perceived stress, depressive symptoms, and maternal parenting behavior are related across the postpartum period. Results indicated large, positive between- and within-person correlations between perceived stress and depression. Neither perceived stress nor depressive symptoms were found to have significant between- or within-person associations with the parenting variables. The second analysis approach utilized an autoregressive cross-lagged model with tests of mediation to identify underlying mechanisms among perceived stress, postpartum depressive symptoms, and maternal parenting behavior in the prediction of infant social-emotional and cognitive development. Results indicated that increased depressive symptoms at 12- and 18-weeks were associated with subsequent reports of increased perceived stress at 18- and 24-weeks, respectively. Perceived stress at 12-weeks was found to be negatively associated with subsequent non-hostility at 18-weeks, and both sensitivity and non-hostility were found to be associated with infant cognitive development and social-emotional competencies at 12 months of age (52-weeks), but not with social-emotional problems. The results of the mediation analyses showed that non-hostility at 18- and 24-weeks significantly mediated the association between perceived stress at 12-weeks and infant cognitive development and social-emotional competencies at 52-weeks. The findings extend research that sensitive parenting in early childhood is as important to the development of cognitive ability, social behavior, and emotion regulation in ethnic minority cultures as it is in majority culture families; that maternal perceptions of stress may spillover into parenting behavior, resulting in increased hostility and negatively influencing infant cognitive and social-emotional development; and that symptoms of depressed mood may influence the experience of stress.
ContributorsCiciolla, Lucia (Author) / Crnic, Keith A (Thesis advisor) / West, Stephen G. (Thesis advisor) / Luecken, Linda J. (Committee member) / Presson, Clark C. (Committee member) / Arizona State University (Publisher)
Created2014
Description
Maternal intrusiveness is an important predictor of child mental health problems. Evidence links high levels of maternal intrusiveness to later infant negativity, and child internalizing problems. However, children also influence the manner in which parents interact with them. For example, infants that show more negative emotionality elicit less positive parenting in their caregivers. Infant affect is also associated with later child internalizing difficulties. Although previous research has demonstrated that maternal intrusiveness is related to infant affect and child internalizing symptomatology, and that infant affect is a predictor of internalizing problems and parenting, no studies have looked at the transactional relations between early maternal intrusiveness and infant affect, and whether these relations in infancy predict later childhood internalizing symptomatology. The present study investigates young children's risk for internalizing problems as a function of the interplay between maternal intrusiveness and infant affect during the early infancy period in a low-income, Mexican-American sample. Participants included 323 Mexican-American women and their infants. Data were collected when the infants were 12, 18, 24, and 52 weeks old. Mothers were asked to interact with their infants in semi-structured tasks, and mother and infant behaviors were coded at 12, 18, and 24 weeks. Maternal intrusiveness was globally rated, and duration of infant negative- and positive affect was recorded. Mother reports of child Internalizing symptomatology were obtained at 52 weeks. Findings suggest that there are transactional relations between early maternal intrusiveness and infant negative affect, while the relations between infant positive affect and maternal intrusiveness are unidirectional, in that infant positivity influences parenting but not vice versa. Further, findings also imply that neither maternal intrusiveness, nor infant affect, influence later toddler internalizing symptomatology. Identifying risk processes in a Mexican-American sample adds to our understanding of emerging infant difficulties in this population, and may have implications for early interventions.
ContributorsRystad, Ida A (Author) / Crnic, Keith A (Thesis advisor) / Enders, Craig (Committee member) / Bradley, Robert (Committee member) / Arizona State University (Publisher)
Created2014
Description
Externalizing behaviors are pervasive, widespread, and disruptive across a multitude of settings and developmental contexts. While the conventional diathesis-stress model typically measures the disordered end of the spectrum, studies that span the range of behavior, from externalizing to competence behaviors, are necessary to see the full picture. To that end, this study examined the additive and nonadditive relations of a dimension of parenting (ranging from warm to rejecting), and variants in dopamine, vasopressin, and neuropeptide-y receptor genes on externalizing/competence in a large sample of predominantly Caucasian twin children in toddlerhood, middle childhood, and early adolescence. Variants within each gene were hypothesized to increase biological susceptibility to both negative and positive environments. Consistent with prediction, warmth related to lower externalizing/higher competence at all ages. Earlier levels of externalizing/competence washed out the effect of parental warmth on future externalizing/competence with the exception of father warmth in toddlerhood marginally predicting change in externalizing/competence from toddlerhood to middle childhood. Warmth was a significant moderator of the heritability of behavior in middle childhood and early adolescence such that behavior was less heritable (mother report) and more heritable (father report) in low warmth environments. Interactions with warmth and the dopamine and vasopressin genes in middle childhood and early adolescence emphasize the moderational role gene variants play in relations between the rearing environment and child behavior. For dopamine, the long variant related to increased sensitivity to parent warmth such that the children displayed more externalizing behaviors when exposed to rejection but they also displayed more competence behaviors when exposed to high warmth. Vasopressin moderation was only present under conditions of parental warmth, not rejection. Interactions with neuropeptide-y and warmth were not significant. The picture that emerges is one of gene-environment interplay, wherein the influence of both parenting and child genotype each depend on the level of the other. As genetic research moves forward, gene variants previously implicated as conferring risk for disorder should be reexamined in conjunction with salient aspects of the environment on the full range of the behavioral outcome of interest.
ContributorsO'Brien, T. Caitlin (Author) / Lemery-Chalfant, Kathryn (Thesis advisor) / Eisenberg, Nancy (Committee member) / Enders, Craig (Committee member) / Nagoshi, Craig (Committee member) / Arizona State University (Publisher)
Created2011
Description
Family adaptation to child developmental disability is a dynamic transactional process that has yet to be tested in a longitudinal, rigorous fashion. In addition, although children with developmental delays frequently have behavior problems, not enough research has examined possible underlying mechanisms in the relation between child developmental delay, adaptation and behavior problems. In the current study, factor analysis examined how best to conceptualize the construct of family adaptation to developmental delay. Also, longitudinal growth curve modeling tested models in which child behavior problems mediated the relation between developmental risk and indices of family adaptation. Participants included 130 typically developing children and their families (Mental Development Index [MDI] > 85) and 104 children with developmental delays and their families (MDI < 85). Data were collected yearly between the ages of three and eight as part of a multi-site, longitudinal investigation examining the interrelations among children's developmental status, family processes, and the emergence of child psychopathology. Results of the current study indicated that adaptation is best conceptualized as a multi-index construct. Different aspects of adaptation changed in unique ways over time, with some facets of adaptation remaining stable while others fluctuated. Child internalizing and externalizing behavior problems were found to decrease over time for both children with developmental delays and typically developing children. Child behavior problems were also found to mediate the relation between developmental risk and family adaptation for over half of the mediation pathways. Significant mediation results indicated that children with developmental delays showed higher early levels of behavior problems, which in turn was associated with more maladaptive adaptation. These findings provide further evidence that families of children with developmental delays experience both positive and more challenging changes in their families over time. This study implies important next steps for research and clinical practice in the area of developmental disability.
ContributorsPedersen y Arbona, Anita (Author) / Crnic, Keith A (Thesis advisor) / Sandler, Irwin (Committee member) / Lemery, Kathryn (Committee member) / Enders, Craig (Committee member) / Arizona State University (Publisher)
Created2011
Description
When people pick up the phone to call a telephone quitline, they are taking an important step towards changing their smoking behavior. The current study investigated the role of a critical cognition in the cessation process--self-efficacy. Self-efficacy is thought to be influential in behavior change processes including those involved in the challenging process of stopping tobacco use. By applying basic principles of self-efficacy theory to smokers utilizing a telephone quitline, this study advanced our understanding of the nature of self-efficacy in a "real-world" cessation setting. Participants received between one and four intervention calls aimed at supporting them through their quit attempt. Concurrent with the initiation of this study, three items (confidence, stress, and urges) were added to the standard telephone protocol and assessed at each call. Two principal sets of hypotheses were tested using a combination of ANCOVAs and multiple regression analyses. The first set of hypotheses explored how self-efficacy and changes in self-efficacy within individuals were associated with cessation outcomes. Most research has found a positive linear relation between self-efficacy and quit outcomes, but this study tested the possibility that excessively high self-efficacy may actually reflect an overconfidence bias, and in some cases be negatively related to cessation outcomes. The second set of hypotheses addressed several smoking-related factors expected to affect self-efficacy. As predicted, higher baseline self-efficacy and increases in self-efficacy were associated with higher rates of quitting. However, contrary to predictions, there was no evidence that overconfidence led to diminished cessation success. Finally, as predicted, shorter duration of quit attempts, shorter time to relapse, and stronger urges all were associated with lower self-efficacy. In conclusion, understanding how self-efficacy and changes in self-efficacy affect and are affected by cessation outcomes is useful for informing both future research and current quitline intervention procedures.
ContributorsGoesling, Jenna (Author) / Barrera, Manuel (Thesis advisor) / Shiota, Lani (Committee member) / Enders, Craig (Committee member) / Presson, Clark (Committee member) / Arizona State University (Publisher)
Created2011
Description
Anxiety sensitivity (AS; the fear of anxiety-related bodily sensations) has been earmarked as a significant risk factor in the development and maintenance of pathological anxiety in adults and children. Given the potential implications of heightened AS, recent research has focused on investigating the etiology and developmental course of elevated AS; however, most of this work has been conducted with adults and is retrospective in nature. Data from college students show that early anxiety-related learning experiences may be a primary source of heightened AS levels, but it remains unclear whether AS in children is linked to their learning experiences (i.e., parental reinforcement, modeling, punishment, and/or transmission of information about anxiety-related behaviors). Based on AS theory and its iterations, an emerging theoretical model was developed to aid further exploration of the putative causes and consequences of heightened AS levels. Using a sample of 70 clinic-referred youth (ages 6 to 16 years old; 51.4% Hispanic/Latino), the present study sought to further explicate the role of learning in the development of AS and anxiety symptoms. Results suggest that childhood learning experiences may be an important precursor to heightened AS levels and, subsequently, increased experiences of anxiety symptoms. Findings also indicate that some youth may be more vulnerable to anxiety-related learning experiences and suggest that culture may play a role in the relations among learning, AS, and anxiety symptoms.
ContributorsHolly, Lindsay (Author) / Pina, Armando A (Thesis advisor) / Crnic, Keith A (Committee member) / Sanabria, Federico (Committee member) / Arizona State University (Publisher)
Created2012
Description
Adolescent substance use carries a considerable public health burden, and early initiation into use is especially problematic. Research has shown that sensation seeking traits increase risk for substance use experimentation, but less is known about individual and contextual factors that can potentially protect against this risk. This study utilized a longitudinal sub sample of youth (N=567) from a larger study of familial alcoholism to examine sensation seeking in early adolescence (ages 10-15) and its relations to later substance use experimentation. Hypotheses tested whether individual executive control, parenting consistency, neighborhood disadvantage, and neighborhood ethnic concentration moderated sensation seeking’s effects on substance use experimentation using multilevel zero-inflated Poisson modeling. Across models, higher levels of sensation seeking were predictive of a higher likelihood of having initiated substance use, but sensation seeking was not significantly related to the number of different substance use classes tried. Only neighborhood disadvantage emerged as a significant moderator of the path from sensation seeking to substance use initiation. The strength of sensation seeking effects on substance use initiation increased as neighborhood disadvantage decreased below average levels, with the most advantaged neighborhoods exhibiting the strongest link between sensation seeking and substance use. There was also a trend towards the most disadvantaged neighborhoods exhibiting increased sensation seeking effects on substance use initiation. These results highlight the importance of focusing on relatively more advantaged areas as potentially risky environments for the externalizing pathway to substance use.
ContributorsJensen, Michaeline R (Author) / Gonzales, Nancy A. (Thesis advisor) / Chassin, Laurie (Thesis advisor) / Enders, Craig (Committee member) / White, Rebecca (Committee member) / Arizona State University (Publisher)
Created2016