Theses and Dissertations
This collection includes both ASU Theses and Dissertations, submitted by graduate students, and the Barrett, Honors College theses submitted by undergraduate students.
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- Creators: Haller, Moira
- Creators: Mauricio, Anne
Description
Posttraumatic Stress Disorder (PTSD) affects nearly 10% of adult women in general population samples. In populations of impoverished ethnic minority women, those lifetime prevalence rates may possibly exceed national averages due to lack of mental health resources. Mothers with PTSD are more likely to exhibit negative parenting styles and experience higher levels of perceived parenting stress, both of which are associated with poor child outcomes. However, there is a lack of evidence on how maternal PTSD may affect parenting for ethnic minority mothers. This study evaluated the prevalence of lifetime PTSD and its effects on parenting stress and infant problem behaviors in a sample of 322 low-income Mexican-American mothers (mean age = 27.8; 86% born in Mexico). Lifetime PTSD diagnoses were assessed at a prenatal home visit (24-36 weeks gestation) using the WHO Composite International Diagnostic Interview (CIDI). Mothers reported parenting hassles at 24-weeks postpartum (PDLH; Crnic & Greenberg, 1990), and child problem behaviors at infant age one-year (BITSEA; Briggs-Gowan et al., 2004). I hypothesized that 1) women with PTSD would report more parenting stress than women without PTSD, 2) women with PTSD would report more infant problem behavior symptoms than women without PTSD, and 3) parenting stress mediates the relationship between PTSD and infant problem behavior. Results found that 16.5% of women met criteria for past or present PTSD. Compared to women without PTSD, women with PTSD reported more parenting stress but a similar level of infant problem behaviors. Parenting stress significantly mediated the relationship between maternal PTSD and infant problem behaviors. Study findings suggest a need for mental health screenings during prenatal care in order to promote the healthy development of high-risk children.
ContributorsPreves, Ashley Maria (Author) / Luecken, Linda (Thesis director) / Davis, Mary (Committee member) / Mauricio, Anne (Committee member) / Department of Psychology (Contributor) / School of Social Transformation (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
Description
The present study utilized longitudinal data from a high-risk community sample (n= 377; 166 trauma-exposed; 54% males; 52% children of alcoholics; 73% non-Hispanic/Latino Caucasian; 22% Hispanic/Latino; 5% other ethnicity) to test a series of hypotheses that may help explain the risk pathways that link traumatic stress, posttraumatic stress disorder (PTSD) symptomatology, and problematic alcohol and drug use. Specifically, this study examined whether pre-trauma substance use problems increase risk for trauma exposure (the high-risk hypothesis) or PTSD symptoms (the susceptibility hypothesis), whether PTSD symptoms increase risk for later alcohol/drug problems (the self-medication hypothesis), and whether the association between PTSD symptoms and alcohol/drug problems is due to shared risk factors (the shared vulnerability hypothesis). This study also examined the roles of gender and ethnicity in these pathways. A series of logistic and negative binomial regressions were performed in a path analysis framework. A composite pre-trauma family adversity variable was formed from measures of family conflict, family life stress, parental alcoholism, and other parent psychopathology. Results provided the strongest support for the self-medication hypothesis, such that PTSD symptoms predicted higher levels of later alcohol and drug problems among non-Hispanic/Latino Caucasian participants, over and above the influences of pre-trauma family adversity, pre-trauma substance use problems, trauma exposure, and demographic variables. Results partially supported the high-risk hypothesis, such that adolescent substance use problems had a marginally significant unique effect on risk for assaultive violence exposure but not on overall risk for trauma exposure. There was no support for the susceptibility hypothesis, as pre-trauma adolescent substance use problems did not significantly influence risk for PTSD diagnosis/symptoms over and above the influence of pre-trauma family adversity. Finally, there was little support for the shared vulnerability hypothesis. Neither trauma exposure nor preexisting family adversity accounted for the link between PTSD symptoms and later substance use problems. These results add to a growing body of literature in support of the self-medication hypothesis. Findings extend previous research by showing that PTSD symptoms may influence the development of alcohol and drug problems over and above the influence of trauma exposure itself, preexisting family risk factors, and baseline levels of substance use.
ContributorsHaller, Moira (Author) / Chassin, Laurie (Thesis advisor) / Davis, Mary (Committee member) / Pina, Armando (Committee member) / Tein, Jenn-Yun (Committee member) / Arizona State University (Publisher)
Created2014