Theses and Dissertations
This collection includes both ASU Theses and Dissertations, submitted by graduate students, and the Barrett, Honors College theses submitted by undergraduate students.
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- Creators: Luecken, Linda
- Creators: Chassin, Laurie
Description
Effortful Control (EC) is a person's ability to self-regulate when presented with an environmental stimulus (Rothbart, et al., 2003). It has been well-established that high levels of EC are associated with multiple positive social and academic outcomes in adolescence (Spinrad et al., 2009). Research suggests that parents have a strong impact on numerous child outcomes, such as EC, through both genetic and environmental pathways. Past research has also examined how parents diagnosed with psychopathology contribute to maladaptive outcomes in their children, including poor regulation, through both genetic and environmental processes (Ellis, et al., 1997). However, less is known about the longitudinal effects of parent dysfunction on the child's environment and regulatory abilities and potential mediators of those effects. The current study tested the hypotheses that parent Alcohol Use Disorder (AUD) would specifically predict early adversity, biological mother conscientiousness, and child EC longitudinally and that early adversity and biological mother conscientiousness would predict child EC. Participants were from a longitudinal study of familial alcoholism (N = 195). Regression analyses indicated that parent AUD was not specifically associated with child EC or with biological mother conscientiousness. However, parent AUD was related to higher levels of early adversity. Additionally, biological mother conscientiousness was associated with higher levels of child EC and early adversity was associated with lower levels of child EC when controlling for earlier EC. Given these findings, future research should test mediation models in which parent AUD predicts child EC indirectly through early adversity.
ContributorsRuof, Ariana Kelsey (Author) / Chassin, Laurie (Thesis director) / Elam, Kit (Committee member) / Davis, Mary (Committee member) / Department of Psychology (Contributor) / Sanford School of Social and Family Dynamics (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
Description
The ability to regulate emotions, attention, and behavior develops early in life and impacts future academic success, social competency, behavioral problems, and psychopathology. An impairment in regulation is known as dysregulation. Past research shows that children of mothers with postpartum depression are more likely to show impairment in regulatory abilities. There is an established link in the literature between family support and maternal depression, which in turn can impact child behavior. However, further research is needed to explore the impact of family support on early childhood dysregulation in the context of maternal depression. Using a sample of 322 Mexican-American, mother-child dyads, two models were examined. Model one hypothesized family support would buffer the effects of maternal depression on child dysregulation at 24 months. Model 2 hypothesized that family support is related to child dysregulation through its effect on maternal depression. Results showed that increased family support was related to more child dysregulation when there were high levels of maternal depression. There was no evidence to support the hypothesis that maternal depression mediated the relationship between family support and child dysregulation.
ContributorsRodrigues, Samantha Jean (Author) / Luecken, Linda (Thesis director) / Benitez, Viridiana (Committee member) / Davis, Mary (Committee member) / Department of Psychology (Contributor) / Sanford School of Social and Family Dynamics (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
Description
Prior research has identified that clinicians in the treatment of eating disorders often do not adhere closely to empirically-supported treatments (EST), and are particularly likely to modify Cognitive-Behavioral therapy (CBT). Several reasons for this phenomenon, dubbed "clinician drift", have been identified, including level of clinician training, education, and type of patient care. In addition to the phenomenon of clinician drift, there has been a growing controversy within the field of clinical psychology about the compatibility of ESTs and multiculturalism. Some argue that the standardization inherent to EST resists the concept of cultural adaptability; while others have countered that cultural adaptability is essential in order for empirically supported treatments to remain relevant, ethical, and effective. In order to shed more light on this issue, this study examined how clinicians tend to drift from CBT in the treatment of Latinos suffering from eating disorders, in order to accommodate Latino culture and elements of eating behavior specific to Latino populations. We both attempted to replicate prior findings regarding predictors of clinician drift, as well as build upon the little existing research into the "culturally-motivated clinician drift." It was discovered that no therapist characteristics or client characteristics were predictive of drift. However, the majority of the sample still adapted or abandoned at least part of the CBT treatment. Their responses regarding the weaknesses of CBT for their Spanish-speaking clients can provide insight into how the treatment can be modified for more diverse clients.
ContributorsJosephs, Jamie Elise (Author) / Perez, Marisol (Thesis director) / Luecken, Linda (Committee member) / Davis, Mary (Committee member) / Department of English (Contributor) / Department of Psychology (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
Description
Posttraumatic Stress Disorder (PTSD) affects nearly 10% of adult women in general population samples. In populations of impoverished ethnic minority women, those lifetime prevalence rates may possibly exceed national averages due to lack of mental health resources. Mothers with PTSD are more likely to exhibit negative parenting styles and experience higher levels of perceived parenting stress, both of which are associated with poor child outcomes. However, there is a lack of evidence on how maternal PTSD may affect parenting for ethnic minority mothers. This study evaluated the prevalence of lifetime PTSD and its effects on parenting stress and infant problem behaviors in a sample of 322 low-income Mexican-American mothers (mean age = 27.8; 86% born in Mexico). Lifetime PTSD diagnoses were assessed at a prenatal home visit (24-36 weeks gestation) using the WHO Composite International Diagnostic Interview (CIDI). Mothers reported parenting hassles at 24-weeks postpartum (PDLH; Crnic & Greenberg, 1990), and child problem behaviors at infant age one-year (BITSEA; Briggs-Gowan et al., 2004). I hypothesized that 1) women with PTSD would report more parenting stress than women without PTSD, 2) women with PTSD would report more infant problem behavior symptoms than women without PTSD, and 3) parenting stress mediates the relationship between PTSD and infant problem behavior. Results found that 16.5% of women met criteria for past or present PTSD. Compared to women without PTSD, women with PTSD reported more parenting stress but a similar level of infant problem behaviors. Parenting stress significantly mediated the relationship between maternal PTSD and infant problem behaviors. Study findings suggest a need for mental health screenings during prenatal care in order to promote the healthy development of high-risk children.
ContributorsPreves, Ashley Maria (Author) / Luecken, Linda (Thesis director) / Davis, Mary (Committee member) / Mauricio, Anne (Committee member) / Department of Psychology (Contributor) / School of Social Transformation (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
Description
The present study utilized longitudinal data from a high-risk community sample (n= 377; 166 trauma-exposed; 54% males; 52% children of alcoholics; 73% non-Hispanic/Latino Caucasian; 22% Hispanic/Latino; 5% other ethnicity) to test a series of hypotheses that may help explain the risk pathways that link traumatic stress, posttraumatic stress disorder (PTSD) symptomatology, and problematic alcohol and drug use. Specifically, this study examined whether pre-trauma substance use problems increase risk for trauma exposure (the high-risk hypothesis) or PTSD symptoms (the susceptibility hypothesis), whether PTSD symptoms increase risk for later alcohol/drug problems (the self-medication hypothesis), and whether the association between PTSD symptoms and alcohol/drug problems is due to shared risk factors (the shared vulnerability hypothesis). This study also examined the roles of gender and ethnicity in these pathways. A series of logistic and negative binomial regressions were performed in a path analysis framework. A composite pre-trauma family adversity variable was formed from measures of family conflict, family life stress, parental alcoholism, and other parent psychopathology. Results provided the strongest support for the self-medication hypothesis, such that PTSD symptoms predicted higher levels of later alcohol and drug problems among non-Hispanic/Latino Caucasian participants, over and above the influences of pre-trauma family adversity, pre-trauma substance use problems, trauma exposure, and demographic variables. Results partially supported the high-risk hypothesis, such that adolescent substance use problems had a marginally significant unique effect on risk for assaultive violence exposure but not on overall risk for trauma exposure. There was no support for the susceptibility hypothesis, as pre-trauma adolescent substance use problems did not significantly influence risk for PTSD diagnosis/symptoms over and above the influence of pre-trauma family adversity. Finally, there was little support for the shared vulnerability hypothesis. Neither trauma exposure nor preexisting family adversity accounted for the link between PTSD symptoms and later substance use problems. These results add to a growing body of literature in support of the self-medication hypothesis. Findings extend previous research by showing that PTSD symptoms may influence the development of alcohol and drug problems over and above the influence of trauma exposure itself, preexisting family risk factors, and baseline levels of substance use.
ContributorsHaller, Moira (Author) / Chassin, Laurie (Thesis advisor) / Davis, Mary (Committee member) / Pina, Armando (Committee member) / Tein, Jenn-Yun (Committee member) / Arizona State University (Publisher)
Created2014
Description
Cardiovascular disease is the leading cause of death in the United States, and classic risk factors only predict half of the variance of cases. In this study, parental overprotection and temperamental negative affectivity both significantly correlated with blood pressure and heart rate, which suggests the importance of examining early life factors when determining one's risk for CVD.
ContributorsCarter, Steven Cross (Author) / Luecken, Linda (Thesis director) / Presson, Clark (Committee member) / Davis, Mary (Committee member) / Barrett, The Honors College (Contributor) / Department of Psychology (Contributor) / School of Life Sciences (Contributor)
Created2013-05
Description
Pediatric chronic pain is pervasive and associated with myriad adverse consequences, yet due consideration has not been given to the mental health disturbances that often present alongside chronic pain and the etiological mechanisms that potentially underlie both. The current study examined the etiology underlying chronic pain and internalizing symptomology in middle childhood, considering both independent and co-occurring symptom presentations. Phenotypic parent-offspring associations across chronic pain and internalizing symptomology were also examined. Lastly, nuclear twin family models were tested to determine the extent to which genetic and environmental factors underlie parent-offspring transmission. The sample comprised 795 children (399 families; Mage= 9.7 years; SD = 0.92) and their parents drawn from the Arizona Twin Project. Results indicated that chronic pain was highly heritable (78%), whereas internalizing symptomology was modestly heritable (32%) and further subject to moderate shared environmental influence (50%). Moreover, 9% of the variance in chronic pain was explained by additive genetic factors shared with internalizing symptomology. Maternal chronic pain and internalizing symptomology were positively associated with both child chronic pain and internalizing symptomology. The association between maternal chronic pain and child chronic pain was more pronounced for girls than boys, whereas the association between maternal internalizing symptomology and child internalizing symptomology was more pronounced for boys than girls. Paternal chronic pain was not significantly associated with child chronic pain but was unexpectedly associated with lower child internalizing symptomology. The negative association between paternal chronic pain and child internalizing symptomology was more pronounced for boys than girls. Paternal internalizing symptomology was not significantly associated with child chronic pain but was positively associated with child internalizing symptomology. Lastly, the best fitting reduced nuclear twin family models for both chronic pain and internalizing symptomology retained additive genetic, sibling-specific shared environmental, and nonshared environmental parameters, where parent-offspring transmission was solely explained by shared genetics and sibling-specific shared environmental factors further accounted for co-twin resemblance. Results provide novel insight into common liabilities underlying chronic pain and internalizing symptomology in middle childhood, parent-offspring associations across chronic pain and internalizing symptomology, and the etiological mechanisms that explain symptom aggregation across generations.
ContributorsOro, Veronica (Author) / Lemery-Chalfant, Kathryn (Thesis advisor) / Chassin, Laurie (Committee member) / Davis, Mary (Committee member) / Su, Jinni (Committee member) / Arizona State University (Publisher)
Created2021