Theses and Dissertations
This collection includes both ASU Theses and Dissertations, submitted by graduate students, and the Barrett, Honors College theses submitted by undergraduate students.
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- Creators: Luecken, Linda
Description
The main objective of this study was to use a genetically-informative design to examine the putative influences of maternal perceived prenatal stress, obstetrical complications, and gestational age on infant dysregulation, competence, and developmental maturity. Specifically, whether or not prenatal and obstetrical environmental conditions modified the heritability of infant outcomes was examined. A total of 291 mothers were interviewed when their twin infants were 12 months of age. Pregnancy and twin birth medical records were obtained to code obstetrical data. Utilizing behavioral genetic models, results indicated maternal perceived prenatal stress moderated genetic and environmental influences on developmental maturity whereas obstetrical complications moderated shared environmental influences on infant competence and nonshared environmental influences on developmental maturity. Gestational age moderated the heritability and nonshared environment of infant dysregulation, shared and nonshared environmental influences on competence, and nonshared environmental influences on developmental maturity. Taken together, prenatal and obstetric conditions were important nonlinear influences on infant outcomes. An evolutionary perspective may provide a framework for these findings, such that the prenatal environment programs the fetus to be adaptive to current environmental contexts. Specifically, prenatal stress governs gene expression through epigenetic processes. Findings highlight the utility of a genetically informative design for elucidating the role of prenatal and obstetric conditions in the etiology of infant developmental outcomes.
ContributorsMcDonald, Kristy (Author) / Lemery-Chalfant, Kathryn S (Thesis advisor) / Fabricius, William (Committee member) / Luecken, Linda (Committee member) / Spinrad, Tracy (Committee member) / Arizona State University (Publisher)
Created2011
Description
The ability to regulate emotions, attention, and behavior develops early in life and impacts future academic success, social competency, behavioral problems, and psychopathology. An impairment in regulation is known as dysregulation. Past research shows that children of mothers with postpartum depression are more likely to show impairment in regulatory abilities. There is an established link in the literature between family support and maternal depression, which in turn can impact child behavior. However, further research is needed to explore the impact of family support on early childhood dysregulation in the context of maternal depression. Using a sample of 322 Mexican-American, mother-child dyads, two models were examined. Model one hypothesized family support would buffer the effects of maternal depression on child dysregulation at 24 months. Model 2 hypothesized that family support is related to child dysregulation through its effect on maternal depression. Results showed that increased family support was related to more child dysregulation when there were high levels of maternal depression. There was no evidence to support the hypothesis that maternal depression mediated the relationship between family support and child dysregulation.
ContributorsRodrigues, Samantha Jean (Author) / Luecken, Linda (Thesis director) / Benitez, Viridiana (Committee member) / Davis, Mary (Committee member) / Department of Psychology (Contributor) / Sanford School of Social and Family Dynamics (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
Description
Prior research has identified that clinicians in the treatment of eating disorders often do not adhere closely to empirically-supported treatments (EST), and are particularly likely to modify Cognitive-Behavioral therapy (CBT). Several reasons for this phenomenon, dubbed "clinician drift", have been identified, including level of clinician training, education, and type of patient care. In addition to the phenomenon of clinician drift, there has been a growing controversy within the field of clinical psychology about the compatibility of ESTs and multiculturalism. Some argue that the standardization inherent to EST resists the concept of cultural adaptability; while others have countered that cultural adaptability is essential in order for empirically supported treatments to remain relevant, ethical, and effective. In order to shed more light on this issue, this study examined how clinicians tend to drift from CBT in the treatment of Latinos suffering from eating disorders, in order to accommodate Latino culture and elements of eating behavior specific to Latino populations. We both attempted to replicate prior findings regarding predictors of clinician drift, as well as build upon the little existing research into the "culturally-motivated clinician drift." It was discovered that no therapist characteristics or client characteristics were predictive of drift. However, the majority of the sample still adapted or abandoned at least part of the CBT treatment. Their responses regarding the weaknesses of CBT for their Spanish-speaking clients can provide insight into how the treatment can be modified for more diverse clients.
ContributorsJosephs, Jamie Elise (Author) / Perez, Marisol (Thesis director) / Luecken, Linda (Committee member) / Davis, Mary (Committee member) / Department of English (Contributor) / Department of Psychology (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
Description
Posttraumatic Stress Disorder (PTSD) affects nearly 10% of adult women in general population samples. In populations of impoverished ethnic minority women, those lifetime prevalence rates may possibly exceed national averages due to lack of mental health resources. Mothers with PTSD are more likely to exhibit negative parenting styles and experience higher levels of perceived parenting stress, both of which are associated with poor child outcomes. However, there is a lack of evidence on how maternal PTSD may affect parenting for ethnic minority mothers. This study evaluated the prevalence of lifetime PTSD and its effects on parenting stress and infant problem behaviors in a sample of 322 low-income Mexican-American mothers (mean age = 27.8; 86% born in Mexico). Lifetime PTSD diagnoses were assessed at a prenatal home visit (24-36 weeks gestation) using the WHO Composite International Diagnostic Interview (CIDI). Mothers reported parenting hassles at 24-weeks postpartum (PDLH; Crnic & Greenberg, 1990), and child problem behaviors at infant age one-year (BITSEA; Briggs-Gowan et al., 2004). I hypothesized that 1) women with PTSD would report more parenting stress than women without PTSD, 2) women with PTSD would report more infant problem behavior symptoms than women without PTSD, and 3) parenting stress mediates the relationship between PTSD and infant problem behavior. Results found that 16.5% of women met criteria for past or present PTSD. Compared to women without PTSD, women with PTSD reported more parenting stress but a similar level of infant problem behaviors. Parenting stress significantly mediated the relationship between maternal PTSD and infant problem behaviors. Study findings suggest a need for mental health screenings during prenatal care in order to promote the healthy development of high-risk children.
ContributorsPreves, Ashley Maria (Author) / Luecken, Linda (Thesis director) / Davis, Mary (Committee member) / Mauricio, Anne (Committee member) / Department of Psychology (Contributor) / School of Social Transformation (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
Description
Research suggests that early family relationships have critical influences on later physical and psychological health, but most studies have focused on the influence of mothers ignoring the unique impacts of fathers. One mechanism by which families may transmit risk is by repeated activation of the hypothalamic-pituitary-adrenal (HPA) axis in the short-term that leads to adult neurobiological dysregulaton, evident in hyper- or hypo-cortisol levels. Using 218 father-child dyads from the Parent and Youth Study (PAYS), the current study investigated whether father involvement in adolescence predicted youth cortisol AUCg and reactivity to a stress task in young adulthood, and whether this relation was mediated by youth perceptions of mattering to their fathers in adolescence. Results revealed that higher father-reported father involvement predicted lower cortisol AUCg in youth when mattering was included in the model, although father involvement was not a statistically significant predictor of AUCg or cortisol reactivity when mattering was not included. Additionally, children who reported higher father involvement also reported higher feelings of mattering, but this association was only statistically significant for girls and European American youth. Youth feelings of mattering did not predict their cortisol reactivity or AUCg in young adulthood. Results suggest that future research should include fathers when investigating the effects of family relationships on youth psychophysiological development.
ContributorsHanna, Mariam A (Author) / Luecken, Linda (Thesis advisor) / Wolchik, Sharlene (Committee member) / Fabricius, William (Committee member) / Arizona State University (Publisher)
Created2015
Description
Cardiovascular disease is the leading cause of death in the United States, and classic risk factors only predict half of the variance of cases. In this study, parental overprotection and temperamental negative affectivity both significantly correlated with blood pressure and heart rate, which suggests the importance of examining early life factors when determining one's risk for CVD.
ContributorsCarter, Steven Cross (Author) / Luecken, Linda (Thesis director) / Presson, Clark (Committee member) / Davis, Mary (Committee member) / Barrett, The Honors College (Contributor) / Department of Psychology (Contributor) / School of Life Sciences (Contributor)
Created2013-05