Theses and Dissertations
This collection includes both ASU Theses and Dissertations, submitted by graduate students, and the Barrett, Honors College theses submitted by undergraduate students.
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- All Subjects: Twin Study
Description
The purpose of the current study was to determine the genetic and environmental contributions to the development of prosocial behavior in children using a population of 356 twins at 8 years of age. The study also aimed to examine whether qualities of parenting (specifically authoritarian and authoritative parenting styles) were phenotypic predictors of prosocial behavior. Both parent-reports and objective ratings of global prosocial behavior were used. Results supported prosocial behavior as a genetically-influenced trait with heritability estimates of 44% and 68% for parent reported and observed prosocial behavior, respectively. Data also suggested prosocial behavior as an environmentally-influenced trait. As hypothesized, authoritative parenting was moderately correlated with parent-reported prosocial behavior and authoritarian parenting was found to be low-to-moderately negatively correlated with parent-reported prosocial behavior. Multi-variable regressions demonstrated that authoritative parenting was significantly predictive of increased parent-reported prosocial behavior but authoritarian parenting was not predictive of decreased parent reported prosocial behavior. However, observed prosocial behaviors were largely independent of both authoritative and authoritarian styles of parenting.
ContributorsLe, Cindy (Author) / Doane, Leah (Thesis director) / Lemery-Chalfant, Kathryn (Committee member) / School of Life Sciences (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
Description
Pediatric chronic pain is pervasive and associated with myriad adverse consequences, yet due consideration has not been given to the mental health disturbances that often present alongside chronic pain and the etiological mechanisms that potentially underlie both. The current study examined the etiology underlying chronic pain and internalizing symptomology in middle childhood, considering both independent and co-occurring symptom presentations. Phenotypic parent-offspring associations across chronic pain and internalizing symptomology were also examined. Lastly, nuclear twin family models were tested to determine the extent to which genetic and environmental factors underlie parent-offspring transmission. The sample comprised 795 children (399 families; Mage= 9.7 years; SD = 0.92) and their parents drawn from the Arizona Twin Project. Results indicated that chronic pain was highly heritable (78%), whereas internalizing symptomology was modestly heritable (32%) and further subject to moderate shared environmental influence (50%). Moreover, 9% of the variance in chronic pain was explained by additive genetic factors shared with internalizing symptomology. Maternal chronic pain and internalizing symptomology were positively associated with both child chronic pain and internalizing symptomology. The association between maternal chronic pain and child chronic pain was more pronounced for girls than boys, whereas the association between maternal internalizing symptomology and child internalizing symptomology was more pronounced for boys than girls. Paternal chronic pain was not significantly associated with child chronic pain but was unexpectedly associated with lower child internalizing symptomology. The negative association between paternal chronic pain and child internalizing symptomology was more pronounced for boys than girls. Paternal internalizing symptomology was not significantly associated with child chronic pain but was positively associated with child internalizing symptomology. Lastly, the best fitting reduced nuclear twin family models for both chronic pain and internalizing symptomology retained additive genetic, sibling-specific shared environmental, and nonshared environmental parameters, where parent-offspring transmission was solely explained by shared genetics and sibling-specific shared environmental factors further accounted for co-twin resemblance. Results provide novel insight into common liabilities underlying chronic pain and internalizing symptomology in middle childhood, parent-offspring associations across chronic pain and internalizing symptomology, and the etiological mechanisms that explain symptom aggregation across generations.
ContributorsOro, Veronica (Author) / Lemery-Chalfant, Kathryn (Thesis advisor) / Chassin, Laurie (Committee member) / Davis, Mary (Committee member) / Su, Jinni (Committee member) / Arizona State University (Publisher)
Created2021