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Description
Various physical and psychological forms of development take place during the transition from childhood to adolescence, including the onset of puberty, delayed sleep-wake behavior patterns, and increases in internalizing symptomatology. Theory suggests that pubertal processes influence the onset of internalizing symptoms, and this association may differ between boys and girls. The contextual amplification hypothesis suggests that pubertal development interacts with contextual or dispositional factors to impact risk for psychopathology. Family stress and peer stress are two critical factors during early adolescence that have potential moderating effects on the association between pubertal development and internalizing symptoms. In line with the biopsychosocial framework, the current study examined various psychosocial (e.g., family stress, peer stress) and biological (e.g., sleep, genetics) factors that may have effects on the puberty-internalizing relation. Participants were a racially/ethnically and socioeconomically diverse sample of twins who were part of an ongoing longitudinal study (N=818 children; Arizona Twin Project; Lemery-Chalfant et al. 2019). The current study examined the association between puberty and internalizing symptoms, with stress (i.e., family, peer) as potential moderators, and sleep indicators (i.e., objective and self-reported sleep) as potential mediators. Behavior genetic analyses explored the moderated heritability of family stress on internalizing symptoms. Findings revealed that greater pubertal development predicted higher internalizing symptoms in boys, but not girls. For girls, peer stress interacted with pubertal development to predict internalizing symptoms, but simple slopes were not significant. Sleep indicators were not significant mediators between pubertal development and internalizing symptoms for boys or girls. Univariate twin models revealed environmental influences on internalizing symptoms, and both genetic and environmental influences on peer stress. Family stress did not significantly moderate the genetic and environmental influences of internalizing symptoms.
ContributorsLecarie, Emma (Author) / Doane, Leah D (Thesis advisor) / Lemery-Chalfant, Kathryn (Committee member) / Gewirtz, Abigail H (Committee member) / Grimm, Kevin J (Committee member) / Arizona State University (Publisher)
Created2024
Description
With the growing popularity of medical cannabis, and high rates of cannabis use disorder (CUD) among medical cannabis users, it is more important than ever to accurately identify the proximal antecedents and subjective effects of medical cannabis use. Subjective antecedents and effects have been proposed as key mechanisms underlying the transition from cannabis use to CUD, but little research has examined medical cannabis users’ experiences in real-time, real-world settings. The current study of 86 young-adult medical cannabis users ages 18-30 (32.6% female) used ecological momentary assessment (EMA) to characterize the antecedents and effects of medical cannabis use, and to examine whether these antecedents and effects vary as a function of CUD severity. Higher craving, pain, and withdrawal symptoms predicted greater odds of cannabis use at the next report, and lower subjective ‘high’ predicted greater odds of cannabis use at the next report. Use of medical cannabis was associated with increases in positive affect, stimulation, relaxation, and subjective ‘high’, decreases in negative affect, withdrawal symptoms, craving, and pain, and increases in cognitive problems, psychotic-like experiences, and adverse bodily effects. Further, following cannabis use, medical users with more CUD symptoms reported greater relief from craving, attenuated increases in stimulation and relaxation, and enhanced increases in sluggishness, cognitive problems, psychotic-like symptoms, and bodily symptoms. Results suggest that medical cannabis use, like recreational use, is associated with a wide range of subjective antecedents and effects, and that relief from cannabis craving may play an important role in the maintenance of CUD among medical users.
ContributorsJones, Connor (Author) / Meier, Madeline H (Thesis advisor) / Chassin, Laurie (Committee member) / Grimm, Kevin J (Committee member) / Corbin, William (Committee member) / Arizona State University (Publisher)
Created2022
Description
The hypothalamic pituitary adrenal (HPA) axis is a primary neuroendocrine system posited to mediate the associations between early life stress and long-term deleterious psychological and physical health outcomes. The effects of early life adversity on HPA axis functioning have been well-documented in primarily White samples, with statistical advances allowing researchers to isolate latent trait cortisol as a stable indicator of HPA axis functioning to account for day-to-day influences on diurnal cortisol patterns. However, directional associations have been mixed depending on developmental stage, demographic composition, and methodological differences across studies. The few studies of early adversity and HPA axis functioning in Hispanic/Latino/a/x samples demonstrate complex interactions between cultural processes and adversity in predicting HPA axis output. Further, nascent literature has isolated the cognitive, meaning-making, and prosocial skills involved in ethnic racial identity (ERI) and its subconstructs of exploration, resolution, and affirmation as promotive during the adolescent stage of development in Latinx youth. Such skills might better prepare youth for neurobiological stress regulation after adversity. To my knowledge, no study has examined whether ERI plays a protective role against the effects of early adversity on trait-level indicators of the HPA axis during adolescence, despite the particularly high rates of cumulative exposure to early life adversity in Latinx youth as compared to White counterparts. Guided by adaptive cultural resilience theories, this study of 197 socioeconomically diverse Latinx older-adolescents aimed to leverage recent findings of stable trait indicators of cortisol output to 1) identify consistent directional markers of the effects of early life adversity on latent trait cortisol in a Latinx sample and 2) elucidate the degree to which ERI might act as a promotive feature for HPA axis levels and protective factor against cumulative early life adversity. Confirmatory factor analyses identified a theory-driven model as an adequate measure of latent trait cortisol. Greater exposure to early adversity predicted lower latent trait cortisol, but ERI demonstrated neither protective nor promotive effects. The present study reifies that early adversity exposure has deleterious effects on trait-level HPA axis functioning, but identifying sources of cultural resilience among Latinx youth remains critical for the future of health equity.
ContributorsGusman, Michaela S. (Author) / Doane, Leah D (Thesis advisor) / Causadias, José M (Committee member) / Grimm, Kevin J (Committee member) / Wolchik, Sharlene A (Committee member) / Arizona State University (Publisher)
Created2022
Description
Drawing upon the theoretical framework of Cooley’s (1902) “looking-glass self” model, the current study examined how affective dimensions of parenting in adolescence contribute to psychopathology in early adulthood through the mediating mechanism of authenticity – one’s level of comfort with being oneself. Using latent profile analysis (LPA), a three-class solution was identified, classifying inadequate, adequate, and optimal profiles of parenting in adolescence. Class membership was used in a multilevel mediation structural equation model to examine longitudinal links with authenticity and psychopathology (e.g., internalizing, externalizing, and substance abuse disorders) in early adulthood. Results demonstrated that optimal compared to inadequate parent-adolescent relationship quality was directly linked to higher levels of authenticity, which in turn, was directly linked to lower levels of all forms of psychopathology in early adulthood. Results also indicated that authenticity fully mediated the link between profiles of parent-adolescent relationship quality (e.g., grade 12) and internalizing, externalizing, and substance abuse disorders in early adulthood (e.g., four years post-college). In conclusion, the current study demonstrated the influence of affective dimensions of parenting profiles in adolescence on the development of psychopathology in early adulthood via the mediating mechanism of authenticity. Moreover, findings from the current study suggest that authenticity is a critical feature shared in common among various forms of psychopathology. Finally, clinical implications are discussed regarding the potential effectiveness of evidence-based psychotherapies aimed at the promotion of authenticity as a mechanism for improving mental health and well-being.
ContributorsEbbert, Ashley Marie (Author) / Infurna, Frank J (Thesis advisor) / Luthar, Suniya S (Committee member) / Grimm, Kevin J (Committee member) / Krasnow, Aaron D (Committee member) / Arizona State University (Publisher)
Created2021
Description
Alcohol use remains a major public health concern and economic burden. Extant literature suggests that young adulthood is a particularly high-risk developmental period for heavy drinking. Given this, it is imperative to understand possible risk and protective factors for heavy drinking and related consequences during this risky developmental period. Prior research has shown that both drinking history and alcohol response (AR) are consistent predictors of future drinking outcomes. However, it is unclear how they may work together to confer this risk. The current study aimed to fill this gap in the literature by examining how alcohol use trajectories across adolescence and early adulthood impacted relations between AR after an alcohol challenge and drinking outcomes over a 2-year period in a sample of young adult moderate to heavy drinkers. Results showed that both drinking history and AR were independently predictive of alcohol outcomes at the 6-month follow-up such that a more extensive drinking history, greater high arousal positive effects, and lesser low arousal negative effects predicted greater drinking and alcohol-related problems 6-months later. However, drinking history and AR were largely not predictive of change in drinking outcomes over time. Finally, AR did not mediate the relationship between drinking history and later alcohol-related outcomes. This is the first study to address relations among drinking history, AR, and later drinking outcomes using a longitudinal alcohol challenge design with a full account of early drinking history. Future research would benefit from inclusion of a broad range of drinkers and longer follow-up assessments to better understand the complex pathways of risk from early drinking history and AR to future drinking outcomes. Such efforts may increase the understanding of who is at greatest risk and/or would benefit most from specific intervention programs.
ContributorsHartman, Jessica (Author) / Corbin, William R (Thesis advisor) / Chassin, Laurie (Committee member) / Doane, Leah D (Committee member) / Grimm, Kevin J (Committee member) / Arizona State University (Publisher)
Created2021