Matching Items (474)
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- All Subjects: Guitar music
ContributorsChan, Robbie (Performer) / McCarrel, Kyla (Performer) / Sadownik, Stephanie (Performer) / ASU Library. Music Library (Contributor)
Created2018-04-18
ContributorsMayo, Joshua (Contributor) / Betts, Sarah (Performer) / Cericola, Marco (Performer) / Gonzalez, Nikolas (Performer) / ASU Library. Music Library (Publisher)
Created2021-04-28
Description
Whenever a text is transmitted, or communicated by any means, variations may occur because editors, copyists, and performers are often not careful enough with the source itself. As a result, a flawed text may come to be accepted in good faith through repetition, and may often be preferred over the authentic version because familiarity with the flawed copy has been established. This is certainly the case with regard to Manuel M. Ponce's guitar editions. An inexact edition of a musical work is detrimental to several key components of its performance: musical interpretation, aesthetics, and the original musical concept of the composer. These phenomena may be seen in the case of Manuel Ponce's Suite in D Major for guitar. The single published edition by Peer International Corporation in 1967 with the revision and fingering of Manuel López Ramos contains many copying mistakes and intentional, but unauthorized, changes to the original composition. For the present project, the present writer was able to obtain a little-known copy of the original manuscript of this work, and to document these discrepancies in order to produce a new performance edition that is more closely based on Ponce's original work.
ContributorsReyes Paz, Ricardo (Author) / Koonce, Frank (Thesis advisor) / Solis, Theodore (Committee member) / Rotaru, Catalin (Committee member) / Arizona State University (Publisher)
Created2013
ContributorsDaval, Charles (Performer) / ASU Library. Music Library (Publisher)
Created2018-03-26
ContributorsMayo, Joshua (Performer) / ASU Library. Music Library (Publisher)
Created2021-04-29
ContributorsDominguez, Ramon (Performer) / ASU Library. Music Library (Publisher)
Created2021-04-15
ContributorsWhite, Bill (Performer) / ASU Library. Music Library (Publisher)
Created2021-04-03
ContributorsSanchez, Armand (Performer) / Nordstrom, Nathan (Performer) / Roubison, Ryan (Performer) / ASU Library. Music Library (Publisher)
Created2018-04-13
Description
Particulate matter (PM) air pollution is a known factor to exacerbate cardiopulmonary diseases. We previously demonstrated that PM mediated endothelial injury and barrier disruption via modulation of the endothelial cytoskeleton and cell-cell junctions, while the effects of PM exposure on cell-cell communication and gap junction activity are still unknown. This study is focused on the characterization of PM-mediated endothelial dysfunction via Connexin 43 (Cx43), the most abundant Gap junction protein expressed in lung endothelial cells (ECs). PM exposure induces a time-dependent elevation of Cx43 in human lung ECs, at both mRNA and protein levels. N-acetyl-cysteine (NAC), an ROS scavenger, significantly suppresses PM-induced Cx43 expression. Membrane-associated and ER/ Golgi apparatus Cx43 protein are elevated upon PM challenge. In addition, PM also activates the gap junction activity, indicated by the transportation of green fluorescence dye between two adjacent ECs. Moreover, GAP27, a selective Cx43 channel inhibitor, attenuates PM-reduced human lung EC barrier disruption, measured by trans-endothelial electrical resistance (TER) with an electric cell-substrate impedance sensing system. Moreover, knock-down the expression of Cx43 by its selective siRNA alleviates PM-induced MLC phosphorylation. These results highly suggest that Cx43 plays a key role in PM-mediated endothelial barrier disruption and signal transduction. Cx43 may deputy as a therapeutic target in PM-mediated cardiopulmonary disorders.
ContributorsKheshtchin-Kamel, Nabia (Author) / Welcome, Natalie (Thesis director) / Wang, Ting (Committee member) / College of Integrative Sciences and Arts (Contributor) / Barrett, The Honors College (Contributor)
Created2020-12