Prior research has established a relation between parenting behaviors and symptoms of child psychopathology, and this association may be influenced by both genetic and environmental factors. Gene-environment correlation, or the influence of a child’s genes on the environment they receive, represents one possible mechanism through which genes and environment combine to influence child outcomes. This study examined evocative gene-environment correlation in the relation between parenting and symptoms of child psychopathology in a sample of 676 twins (51.5% female, 58.5% Caucasian, 23.7% Hispanic/Latinx, primarily middle class, MAge=8.43, SD=.62) recruited from Arizona birth records. Using univariate ACE twin biometric models, genetic influences were found to moderately contribute to internalizing symptoms (A=.47, C=.25, E=.28), while externalizing (A=.86, E=.14) and ADHD (A=.84, E=.16) symptoms were found to be highly heritable. The genetic influences for positive (C=.54, E=.46) and negative (C=.44, E=.56) parenting were smaller and found to be nonsignificant. The correlations between parenting and types of psychopathology were examined and bivariate Cholesky decompositions were conducted for statistically significant correlations. Negative parenting was moderately positively correlated with externalizing and ADHD symptoms; the relation between externalizing symptoms and negative parenting was found to be due to shared genetics, whereas the relation between negative parenting and ADHD symptoms was due to the shared environment. The mixed results regarding the role of gene environment correlation in relations between parenting and child psychopathology indicate that further research on the mechanisms of this relation is needed.

Attention-deficit hyperactivity disorder (ADHD) is a developmental disorder categorized by symptoms of inattention, impulsivity, and/or hyperactivity. The onset of ADHD symptoms begin to appear in childhood, tend to be persistent into adulthood, and are associated with adverse physical and mental health outcomes (Caye et al., 2016; Yoshimasu et al., 2018). The current study examined the effects of health-promoting behavior (i.e., diet and physical activity) on ADHD symptoms in middle childhood. The sample consisted of 670 twins (30.1% monozygotic, 36.5% same-sex dizygotic, 32.3% opposite-sex dizygotic) from the ongoing longitudinal study, the Arizona Twin Project. The twin’s (51% female) diet, physical activity, and ADHD symptoms were assessed at age 8 (M = 8.45, SD = 0.69) and ADHD symptoms were assessed again at age 9 (M = 9.70, SD = 0.92). Mixed model regression analyses revealed that aspects of diet (i.e., protein, carbohydrates, and fiber) at age 8 negatively predicted ADHD symptoms at age 9. Similarly, sedentary behavior at age 8 negatively predicted ADHD symptoms at age 9, whereas moderate-to-vigorous activity at age 8 positively predicted ADHD symptoms at age 9. Univariate twin analyses revealed that certain aspects of diet (i.e., sugar, vegetable, and fruit consumption) were influenced by environmental factors whereas other aspects of diet (i.e., protein, carbohydrates, calories, fat, and fiber consumption) were influenced by both genetic and environmental factors. Both children’s sedentary behavior and moderate-to-vigorous activity were influenced primarily by genetic factors, with the remaining variance being attributed to non-shared environmental factors. Additive genetic influences explained the majority of the variance in ADHD symptoms. Future research should examine bidirectional effects of activity and diet on ADHD symptoms across childhood.

The neighborhood context is an important predicator of developmental outcomes, as it is where children spend much of their time. Especially when it comes to urban, low-income areas, high neighbor danger and crime have been considered a developmental risk. Research has documented the links between neighborhood environments and academic achievement, but less is known regarding the holistic view that considers contexts in conjunction with the neighborhood, despite these being important levels of influence. Thus, this study examined: 1) the direct associations between parent report of neighborhood danger and objectively measured neighborhood crime and academic achievement (e.g., Woodcock-Johnson IV), 2) family level stress as a potential mediator in these links, and 3) the teacher-child relationship as a potential moderator of these associations. Participants were from the ethnically and socioeconomically diverse Arizona Twin Project study (N=707 twin children, Mage = 8.44 years; SD = 0.69; 28.0% Hispanic/Latino, 57.7% Non-Hispanic White, 3.4% Asian American, 3.8% African American, 2.6% Native American, and 2.8% multiethnic or other; 53.5% middle to upper class, 15.6% lower middle class, 21.6% living near the poverty line, and 7.4% living in poverty as calculated by an income to needs ratio). There were no direct effects between subjective neighborhood danger and indicators of academic achievement, but there was a positive association between objective crime and scores on applied problems (an indicator of math achievement). Family- level stress was a significant mediator of perceptions of neighborhood danger and scores on passage comprehension (an indicator of reading achievement) but did not play a mediating role in the relation between objective crime and academic achievement. Specifically, higher levels of danger and crime were associated with greater levels of family stress, and lower levels of academic achievement scores. The teacher-child relationship was not a moderator in the relation between neighborhood danger/crime and academic achievement. Study findings provide additional evidence regarding the differences in subjectively versus objectively obtained data on the neighborhood environment and can inform future intervention (e.g., in the home and at schools) that support student’s academic achievement by addressing multiple levels of contextual influence.