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Description
Current research attempts to address the increasing prevalence of Alzheimer’s disease (AD) by
finding causes and treatments to revert misfolded proteins and ceasing progression due to
Diabetes Mellitus (DM). The goal of this review is to highlight the contribution of misfolded Tau
protein to AD through neurofibrillary tangles solely, and in conjunction with known causative
agents such as 𝛽-amyloid protein. Finally, it interprets the association of Tau with DM and its
effects on the progression of AD.
finding causes and treatments to revert misfolded proteins and ceasing progression due to
Diabetes Mellitus (DM). The goal of this review is to highlight the contribution of misfolded Tau
protein to AD through neurofibrillary tangles solely, and in conjunction with known causative
agents such as 𝛽-amyloid protein. Finally, it interprets the association of Tau with DM and its
effects on the progression of AD.
ContributorsTavani, Jennifer Renee (Author) / Houtchens, Jason (Thesis director) / Lisenbee, Cayle (Committee member) / College of Health Solutions (Contributor) / Barrett, The Honors College (Contributor)
Created2020-05