Matching Items (3)
Description
Prior research has established associations between sleep duration and body mass index (BMI) scores and risk for obesity in middle childhood, but it is less clear whether other objectively- and subjectively-measured sleep indicators may be associated with BMI scores, weight status (e.g., obesity), and other estimates of weight and body fat such as waist circumference (WC) and percent body fat. Empirical studies have also demonstrated independent associations between broad self-regulation and sleep indicators and BMI scores, but no study to date has tested these factors in a model together and the extent to which associations between normative sleep problems, weight indicators, and effortful control (EC) may be explained by shared genetic or environmental influences. Data from a large longitudinal study of twins was used to test phenotypic associations between sleep problems at eight years and weight indicators at nine years, including whether EC at eight years moderates these associations. Additionally, multiple quantitative behavior genetic models were used to estimate unique and shared genetic and environmental covariances among normative sleep problems, weight indicators, and EC at eight years of age and whether additive genetic influence on weight in middle childhood differs by child weight status group. Phenotypic findings showed that greater sleep duration at eight years predicted greater decreases BMI at nine years of age for children with low levels of EC at eight years. Greater sleep midpoint variability at eight years predicted greater increases in percent body fat from eight to nine years of age for children with low EC at eight years. Behavior genetic findings showed greater environmental influences on parent-reported sleep duration and quality, as well as objective sleep midpoint variability. Similarly, associations between parent-reported sleep duration and sleep midpoint variability and other sleep indicators and EC were primarily accounted for by shared environmental factors. In contrast, there was high additive genetic influence on objective sleep quantity and quality, all weight indicators, and EC. Many of the associations between sleep indicators, sleep and weight indicators, and among weight indicators were entirely accounted for by shared additive genetic factors, suggesting that common, underlying sets of genes explain these relations.
ContributorsBreitenstein, Reagan Styles (Author) / Doane, Leah D. (Thesis advisor) / Lemery-Chalfant, Kathryn (Committee member) / Perez La Mar, Marisol (Committee member) / Grimm, Kevin (Committee member) / Arizona State University (Publisher)
Created2019
Description
Childhood obesity is associated with many well established health risks as well as high annual public health costs. Because of this, the childhood obesity literature has highlighted the need to detect at-risk groups in order to implement targeted preventions. Emotional eating has been identified as an unhealthy behavior and a risk factor for overweight status among children though very little is known about what predisposes children to emotionally eat. Stress has often been found to elicit emotional eating but most studies looking at this relationship have relied on self-reports in adult and clinical samples. Thus, the current study seeks to investigate the relationship between stress reactivity (measured using heart rate variability) and emotional eating in a sample of 247 children between the ages of 4-6. Furthermore, levels of control may moderate the relationship between stress reactivity (HRV) and emotional eating. Linear regression analysis was used to explore these relationships. It was expected that higher levels of reactivity to stress would predict an increased likelihood of emotional eating. This association was expected to be attenuated among children with higher levels of inhibitory control and attentional focusing as well as lower levels of impulsivity. However, the hypothesized findings were not supported by the data. Despite these null findings, and in light of several limitations, it is still hypothesized that emotional eating involves physiological and impulsivity/effortful control processes. Implications of future research are discussed.
ContributorsOhrt, Tara K (Author) / Perez La Mar, Marisol (Thesis advisor) / Luecken, Linda (Committee member) / Lemery, Kathryn (Committee member) / Arizona State University (Publisher)
Created2016
Description
The present study contributed an investigation of prosocial peers, a prospective promotive factor, and its association with depressive symptoms, an internalizing outcome. The study utilized six waves of panel data from 2,002 youth in the control condition of the Community Youth Development Study (mean age 13.12 at Grade 7; 52% male; 66.1% White; 26.6% Hispanic). A series of time-varying effect models (TVEM) illustrated the associations between prosocial peers and depressive symptoms over developmental time from Grades 7 through 12. It was hypothesized that prosocial peers and depressive symptoms would have a negative association for both males and females, and that the association would be moderated by gender at the time of transition to high school. It was expected that females would display a significantly stronger negative association than males between prosocial peers and depressive symptoms at this juncture, particularly due to gender-based differences in socialization that are compounded by transition. To strengthen conclusions about prosocial peers being a promotive factor, secondary analyses included covariates measuring previous levels of depressive symptoms; these models accounted either for baseline depressive symptoms or year-prior symptoms. Results showed, overall, prosocial peers had a significant negative association with depressive symptoms over time, for both males and females. When controlling for baseline depressive symptoms, this was still the case. When controlling for year-prior depressive symptoms, prosocial peers was no longer significantly associated with depressive symptoms for males across Grades 10 through 12. Gender moderated the association between prosocial peers and depressive symptoms at the time of transition to high school as well as other grades. When controlling for baseline depressive symptoms, it was again found that gender moderated the association between prosocial peers and depressive symptoms at the time of transition to high school (Grades 8 and 9) but also at Grade 12. When controlling for year-prior depressive symptoms, gender did not moderate the association at the time of transition to high school, but it did at Grades 10, 11, and 12. Overall, results support the possibility of prosocial peers as a prospective promotive factor for youth mental health.
ContributorsMendes, Skyler H. (Author) / Oesterle, Sabrina (Thesis advisor) / Doane, Leah (Thesis advisor) / Perez La Mar, Marisol (Committee member) / Arizona State University (Publisher)
Created2021