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Description
Children's chronic pain has many contributing factors, including family environment, genetics, and parenting. Still, pediatric chronic pain remains understudied, and little research has been conducted on predictors of child pain onset. This study aims to elucidate some of these factors by examining the role of parenting style and parental pain

Children's chronic pain has many contributing factors, including family environment, genetics, and parenting. Still, pediatric chronic pain remains understudied, and little research has been conducted on predictors of child pain onset. This study aims to elucidate some of these factors by examining the role of parenting style and parental pain in children's chronic pain experience. The study answered the following questions: 1) Is child chronic pain heritable?; 2) Do parenting styles and/or parental pain predict child pain?; and 3) Is parenting style the mediating variable in the relation between parent pain and child pain? A twin study design was employed to account for both genetic and environmental influences in pain. Primary and secondary caregivers completed pain questionnaires regarding their own and their children's pain. The caregivers also completed questionnaires regarding their own parenting styles. Observer ratings were used as additional measures of primary caregiver parenting. Results indicated that child pain is heritable and that parental pain was significantly related to child pain. However, parenting style did not predict child pain and was not a mediator in the relationship between parental pain and child pain. Further research on other parenting factors or predictors of pain may lead to prevention of pediatric chronic pain or more effective management of child pain symptoms.
ContributorsPatel, Maya (Author) / Davis, Mary (Thesis director) / Lemery, Kathryn (Thesis director) / Department of Psychology (Contributor) / Barrett, The Honors College (Contributor)
Created2018-05
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Description
This study addresses a gap in the literature by examining interactions between parental monitoring and parental autonomy giving/personal autonomy in predicting changes in drinking behavior from high school to college. Using data from two unique studies (study 1 was 62.8% female, n = 425; study 2 was 59.9% female, n

This study addresses a gap in the literature by examining interactions between parental monitoring and parental autonomy giving/personal autonomy in predicting changes in drinking behavior from high school to college. Using data from two unique studies (study 1 was 62.8% female, n = 425; study 2 was 59.9% female, n = 2245), we analyzed main effects of parental monitoring, parental autonomy-giving, and personal autonomy. We also analyzed interactions between parental monitoring and autonomy-giving, and between parental monitoring and personal autonomy. Analyses found significant main effects of parental monitoring on drinking, with high levels of parental monitoring protecting against heavy drinking. Personal autonomy was a protective factor in both high school and college, whereas parental autonomy-giving did not predict drinking behavior in either high school or during the transition to college. This calls into question the extent to which parental autonomy-giving is a primary influence on personal autonomy. Hypothesized interactions between parental monitoring and parental autonomy giving/personal autonomy were not statistically significant. In summary, parental monitoring seems to be protective in high school, and personal autonomy—but not parental autonomy-giving—is also protective. Whereas the latter finding is well established from previous studies, the protective effect of personal autonomy during the transition to college is a novel finding. This relationship suggests that efforts to identify sources of personal autonomy in early adulthood and methods for increasing autonomy may be warranted.
ContributorsStack, Jaclyn Elaine (Author) / Corbin, William (Thesis director) / Meier, Madeline (Committee member) / Davis, Mary (Committee member) / Department of Psychology (Contributor) / Barrett, The Honors College (Contributor)
Created2017-12
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Description
Pediatric obesity is a public health concern due to its elevated prevalence rates and its relation to concurrent and long-term physical and psychosocial consequences. Pediatric obesity has been found to be associated with problem behaviors, albeit with inconsistent findings. The mechanism of this relation is unclear. It is possible

Pediatric obesity is a public health concern due to its elevated prevalence rates and its relation to concurrent and long-term physical and psychosocial consequences. Pediatric obesity has been found to be associated with problem behaviors, albeit with inconsistent findings. The mechanism of this relation is unclear. It is possible that they have a shared etiology. Self-regulation and parenting practices are two factors that have been implicated in the development of problem behaviors and are garnering evidence for their relation with pediatric obesity. The goal of the present study was to examine whether self-regulation (SREC), positive behavior support (PBSEC), and coercive limit-setting (CLSEC) in early childhood are shared etiological factors of pediatric obesity and problem behaviors. Using multinomial logistic regression the likelihood of belonging to four outcome groups (Comorbid, Problem behavior only, Overweight only, and Typically developing) at age 10 based on these factors was assessed. Analyses controlled for intervention group assignment, child gender, child African-American or Bi-racial, child Hispanic, cumulative risk, child body size impression at age 2, and parent body size impression at baseline. In the models examining SREC alone, for every 1 standard deviation increase in SREC, there was a reliable reduction in the odds of the child belonging to the comorbid and problem behavior only groups at age 10, compared to the typically developing group (OR = 0.386, 95% CI [0.237, 0.628], OR = 0.281, 95% CI [0.157, 0.503], respectively). This relation was maintained when SREC was in the same model as PBSEC and CLSEC. PBSEC and CLSEC alone did not impact the likelihood of belonging to any of the outcome groups. A significant interaction was found between SREC and CLSEC, such that at high levels of both SREC and CLSEC the odds of a child belonging to the overweight only group at age 10 increased, compared to the typically developing group. Results highlight CLSEC as a parenting practice that may place a highly regulated child at risk for becoming overweight. Overall, the findings suggest that problem behaviors and pediatric obesity do not have a shared etiology.
ContributorsMontano, Zorash (Author) / Dishion, Thomas J (Thesis advisor) / Gonzales, Nancy (Committee member) / Perez, Marisol (Committee member) / Enders, Craig (Committee member) / Arizona State University (Publisher)
Created2016
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Description

Prior research has established a relation between parenting behaviors and symptoms of child psychopathology, and this association may be influenced by both genetic and environmental factors. Gene-environment correlation, or the influence of a child’s genes on the environment they receive, represents one possible mechanism through which genes and environment combine

Prior research has established a relation between parenting behaviors and symptoms of child psychopathology, and this association may be influenced by both genetic and environmental factors. Gene-environment correlation, or the influence of a child’s genes on the environment they receive, represents one possible mechanism through which genes and environment combine to influence child outcomes. This study examined evocative gene-environment correlation in the relation between parenting and symptoms of child psychopathology in a sample of 676 twins (51.5% female, 58.5% Caucasian, 23.7% Hispanic/Latinx, primarily middle class, MAge=8.43, SD=.62) recruited from Arizona birth records. Using univariate ACE twin biometric models, genetic influences were found to moderately contribute to internalizing symptoms (A=.47, C=.25, E=.28), while externalizing (A=.86, E=.14) and ADHD (A=.84, E=.16) symptoms were found to be highly heritable. The genetic influences for positive (C=.54, E=.46) and negative (C=.44, E=.56) parenting were smaller and found to be nonsignificant. The correlations between parenting and types of psychopathology were examined and bivariate Cholesky decompositions were conducted for statistically significant correlations. Negative parenting was moderately positively correlated with externalizing and ADHD symptoms; the relation between externalizing symptoms and negative parenting was found to be due to shared genetics, whereas the relation between negative parenting and ADHD symptoms was due to the shared environment. The mixed results regarding the role of gene environment correlation in relations between parenting and child psychopathology indicate that further research on the mechanisms of this relation is needed.

ContributorsCarrizosa, Mya Grace (Author) / Lemery-Chalfant, Kathryn (Thesis director) / Corbin, William (Committee member) / Davis, Mary (Committee member) / Oro, Veronica (Committee member) / Department of Information Systems (Contributor) / Economics Program in CLAS (Contributor) / Department of Psychology (Contributor) / Barrett, The Honors College (Contributor)
Created2021-05