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- All Subjects: parenting
- Creators: Spinrad, Tracy
- Creators: Chassin, Laurie
(RSA) interaction in predicting these trajectories. Child sex was tested as a covariate and moderator. It was found that children's low baseline RSA or high RSA reactivity , in comparison to high baseline RSA or low RSA reactivity , was more reactive as a function
of early parenting quality when predicting the development of early childhood problem symptoms. Differential patterns of the interaction between parenting quality and RSA were detected for mothers' and fathers' reports. Mother-reported models showed a diathesis-stress pattern, whereas the father-reported model showed a vantage-sensitivity pattern, especially for internalizing symptoms. This may imply the potential benefit of fathers' active engagement in children's early development. In addition, the effect of the parenting quality × RSA interaction in predicting the mother-reported models was found
to be further moderated by child sex. Specifically, the parenting quality × baseline RSA interaction was significantly predictive of girls' 54-month internalizing, and the parenting quality × RSA reactivity interaction significantly predicted boys' internalizing slope. Girls with low baseline RSA or boys with high RSA reactivity were vulnerable to the less positive parenting, exhibiting high levels of 54-month internalizing symptoms or slow decline in internalizing over time, respectively. Future research directions were discussed in terms of integrating the measures of SNS and PNS in psychopathology study,
exploring the mechanisms underlying the sex difference in parenting quality × RSA interaction, and comparing the findings of children's typical and atypical development.
Analyses were performed in a path analysis framework. To test these research questions, the current study employed two polygenic risk scores. The first, a theory-based score, was formed using single-nucleotide polymorphisms (SNPs) from receptor systems implicated in the amplification of positive effects in the presence of new/exciting stimuli and/or pleasure derived from using substances. The second, an empirically-based score, was formed using a data-driven approach that explained a large amount of variance in SUDs. Together, these scores allowed the present study to test explanations for the relations among parent AUD, parental knowledge, peer substance use, and SUDs.
Results of the current study found that having parents with less knowledge or an AUD conferred greater risk for SUDs, but only for those at higher genetic risk for behavioral undercontrol. The current study replicated research findings suggesting that peer substance use mediated the effect of parental AUD on SUD. However, it adds to this literature by suggesting that some mechanism other than increased behavioral undercontrol explains relations among parental AUD, peer substance use, and emerging adult SUD. Taken together, these findings indicate that children of parents with AUDs comprise a particularly risky group, although likelihood of SUD within this group is not uniform. These findings also suggest that some of the most important environmental risk factors for SUDs exert effects that vary across level of genetic propensity.
Child temperamental anger and frustration was found to moderate reciprocal relations across all three parent-to-child cross-lagged paths. Children scoring high on a dispositional proclivity to react with anger and frustration were more likely to avoid maternal hostility, via a significant decrease in negativity, across time. Moderation was also supported in two of three child-to-parent lagged paths. Finally, maternal neuroticism moderated the reciprocal effects during early childhood, such that more neurotic mothers were more likely to demonstrate a decrease in the probability of hostility relative to mothers scoring lower on neuroticism. This affect was attenuated in middle childhood, with patterns becoming similar between mothers scoring high and low on neuroticism. Moreover, children of less neurotic mothers were more likely to demonstrate a decrease in the probability of exhibiting negativity from 36 to 54 months compared to children of more neurotic mothers. This effect also attenuated with patterns becoming negative at the grade 1 to grade 3 lag. Overall, the results from this study supported a transactional model of parent-child relationships, were consistent with the motivation literature, did not support a coercive process of interaction when the sample and measurement paradigm were low-risk, and generally suggested parents and children have an equal influence on the relational processes investigated from early to middle childhood.
Adolescence is an important period of scaffolding for educational attainment, which is among the strongest predictors of outcomes in multiple domains. Parents who encourage academic success and promote self-regulation may enhance their offspring’s educational attainment. However, parents with externalizing disorders present a complex constellation of risk factors, including low educational attainment and poor parenting, and are more likely to have children with high levels of disinhibition. Previous research has identified low parental education, poor parenting and adolescent impulsivity as threats to educational attainment, but has not examined risk factors for discrepancies in educational attainment among siblings of the same family. Furthermore, studies have not examined the between- and within-family mechanisms that may explain why adolescents with externalizing parents have low educational attainment. The current study addressed these gaps by testing between- and within-family predictors of educational attainment using data from a longitudinal, multigenerational study that oversampled families at risk for alcohol use disorder. The sample consisted of 555 biological siblings within 240 families. We tested whether parental externalizing predicted lower educational attainment through parents’ own lower education, parents’ differential treatment of offspring, and impulsivity. Results suggested that between families, parents with externalizing disorders had lower educational attainment and more impulsive offspring, but did not exhibit increased differential parenting. Within families, siblings with greater impulsivity had lower educational attainment, whereas receiving more preferential maternal treatment than one’s siblings predicted higher educational attainment. Low parental educational attainment mediated the relation between parental externalizing disorders and low offspring educational attainment.
In the present study, I will focus on several aspects of parenting (monitoring, structure, positive parenting, harsh discipline) and the relations with social competence. The larger study, on which this paper is based, was intended to study multiple types of parenting behaviors and social competencies, and development of measures was culturally and developmentally informed (including focus groups and pilot collection). However, utilizing each dimension that emerged from analyses of the parenting behaviors and social competence measures would result in a study with too large a scope. I will include each aspect of parenting that emerged in analyses. However, I will focus on just one of the two factors of social competence that emerged in analyses for adolescents. This first factor includes prosocial behavior (helping and sharing; Eisenberg et al., 2006) and also is composed of general social competence items capturing adolescents’ use of manners and politeness. For the purposes of this paper, I will refer to this first factor as “social competence,” and I will draw on the general social competence literature and prosocial behavior literature.
Participants (N = 236 in models that included multiple G2 siblings; N = 110 in models without siblings) were drawn from a larger sample of at-risk (i.e., alcoholic) and comparison families followed longitudinally for over 30 years and across three generations. Four mediation models were estimated to examine effects of two separate G1-G2 family disruption components (deviance-related and health-related disruption) on parenting of G3, mediated by family conflict, family disorganization, and G2 dysregulation. Results indicated that health-related disruption impairs consistency of parenting provided to G3 offspring through conflict in the G1-G2 family. A direct effect of health-related disruption was also seen on parental monitoring. There were no direct or mediated effects of deviance-related disruption on parenting. Implications and future directions will be discussed.