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          <dc:identifier>https://hdl.handle.net/10776/2098</dc:identifier>
                  <dc:date>2011-05-04</dc:date>
                  <dc:language>eng</dc:language>
                  <dc:contributor>O&#039;Neil, Erica</dc:contributor>
          <dc:contributor>Arizona State University. School of Life Sciences. Center for Biology and Society. Embryo Project Encyclopedia.</dc:contributor>
          <dc:contributor>Arizona Board of Regents</dc:contributor>
                  <dc:rights>open access</dc:rights>
          <dc:rights>http://creativecommons.org/licenses/by-nc-sa/3.0/</dc:rights>
                  <dc:description>Maternal consumption of alcohol (ethanol) can result in a range of alcohol-induced developmental defects. In humans, those collective birth defects are called Fetal Alcohol Spectrum Disorders, with the most severe manifestation being Fetal Alcohol Syndrome (FAS). FAS is defined by pre- and post-natal growth retardation, minor facial abnormalities, and deficiencies in the central nervous system (CNS). The eye and ocular system development is particularly susceptible to the effects of prenatal alcohol exposure and can result in visual impairment or blindness.</dc:description>
                  <dc:subject>fetal alcohol syndrome</dc:subject>
          <dc:subject>Reproduction</dc:subject>
          <dc:subject>Human Development</dc:subject>
                  <dc:title>Effects of Prenatal Alcohol Exposure on Ocular Development</dc:title></oai_dc:dc></metadata></record></GetRecord></OAI-PMH>
