2026-05-23T13:30:36Zhttps://keep.lib.asu.edu/oai/requestoai:keep.lib.asu.edu:node-1313992024-12-18T20:58:30Zoai_pmh:alloai_pmh:repo_items131399
https://hdl.handle.net/2286/R.I.56598
http://rightsstatements.org/vocab/InC/1.0/
2020-05
30 pages
eng
Tavani, Jennifer Renee
Houtchens, Jason
Lisenbee, Cayle
College of Health Solutions
Barrett, The Honors College
Text
Current research attempts to address the increasing prevalence of Alzheimer’s disease (AD) by<br/>finding causes and treatments to revert misfolded proteins and ceasing progression due to<br/>Diabetes Mellitus (DM). The goal of this review is to highlight the contribution of misfolded Tau<br/>protein to AD through neurofibrillary tangles solely, and in conjunction with known causative<br/>agents such as 𝛽-amyloid protein. Finally, it interprets the association of Tau with DM and its<br/>effects on the progression of AD.
Tau Protein
Alzheimer's Disease
Type 2 Diabetes
microtubules
Reviewing the Effects of Misfolded Tau Proteins in Alzheimer’s Disease