Description
In cancer, various genetic and epigenetic alterations cause cancer cells to hyperproliferate and to bypass the survival and migration mechanisms that typically regulate healthy cells. The focal adhesion kinase (FAK) gene produces FAK, a protein that has been implicated in tumor progression in various cancers. Compared with normal tissue counterparts, FAK is overexpressed in many cancers.
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Contributors
- Nott, Rohini (Author)
- Gould, Ian R. (Thesis director)
- Marlowe, Timothy A. (Committee member)
- Cance, William G. (Committee member)
- School of Life Sciences (Contributor)
- Dean, W.P. Carey School of Business (Contributor)
- Barrett, The Honors College (Contributor)
Date Created
The date the item was original created (prior to any relationship with the ASU Digital Repositories.)
2019-05
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