Description
Objective: Progressive accumulation of α-synuclein (α-syn) has been associated with Parkinson's disease (PD) and Dementia with Lewy body (DLB). The mechanisms through which α-syn leads to neurodegeneration are not completely clear; however, the formation of various oligomeric species have been proposed to play a role.
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Contributors
- Spencer, Brian (Author)
- Williams, Stephanie (Author)
- Rockenstein, Edward (Author)
- Valera, Elvira (Author)
- Xin, Wei (Author)
- Mante, Michael (Author)
- Florio, Jazmin (Author)
- Adame, Anthony (Author)
- Masliah, Eliezer (Author)
- Sierks, Michael (Author)
- Ira A. Fulton Schools of Engineering (Contributor)
Date Created
The date the item was original created (prior to any relationship with the ASU Digital Repositories.)
2016-06-16
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Identifier
- Digital object identifier: 10.1002/acn3.321
- Identifier TypeInternational standard serial numberIdentifier Value2328-9503
Note
- The final version of this article, as published in Annals of Clinical and Translational Neurology, can be viewed online at: http://onlinelibrary.wiley.com/wol1/doi/10.1002/acn3.321/abstract, opens in a new window
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Spencer, B., Williams, S., Rockenstein, E., Valera, E., Xin, W., Mante, M., . . . Sierks, M. R. (2016). α-synuclein conformational antibodies fused to penetratin are effective in models of Lewy body disease. Annals of Clinical and Translational Neurology, 3(8), 588-606. doi:10.1002/acn3.321